EMF Blocking and even EMF Shielding
No matter if you live in an apartment or house or just want to keep your home free of EMFs, there are a number of methods to reduce exposure. One of the easiest is to reduce your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from reaching your house. Another easy way to protect your home against EMF radiations is to use a shielding canopy for RF. It is a type made of net which contains EMF shielding. It is utilized to prevent EMFs from entering rooms. Another option is to have your house equipped with an electrical enclosure. They are referred to as Faraday cages.
A number of studies have proven how the non-ionizing energy of RF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation of cancer stem cells. This could be the reason for the long-term response seen in some patients with advanced HCC. However, the mechanism behind AM EMF's impact on cancer patients is not yet clear.
Effects on the effects of AM RF EMF on HCC tumour growth in vivo were examined in mice. The tumors were split into 3 groups. The first group was not exposed to RF EMF. The second group was exposed RF EMF at frequencies similar to the one used by humans. The third group was exposed the RF EMF in HCC-specific frequencies. emf blocker of HCCMF on the tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF showed significant shrinkage. However, emf blocking treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism behind tumor-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is mediated through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. emf blocking suggest that CACNA1H could have more broader implications in the diagnosis and treatment of various cancers.
The tumors in those in the group that were unaffected to RF EMF, and were fed a normal diet of mice. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumors were then killed after they had a high burden.
The tumors of the three groups showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumours treated with RCF did not show any reduction in size. The difference was significant. The tumors treated by RCF were able to show necrosis, which is common in tumors that have been exposed to RCF. The possibility is that the necrosis was caused by the lack of oxygen in larger cancers.
In summary, the results show the fact that AM EMF has anticancer properties in vitro as well as in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF causes these effects through CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting impact on the development of HCC tumors in vivo.