EMF Blocking plus EMF Shielding
No matter if you live in an apartment or house or simply want to keep your home free of EMFs There are plenty options to limit your exposure. One of the most effective is to reduce your electronic device use. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiation is to use an RF shielding canopy. This is a cloth made of net which contains EMF shielding. It's used to stop EMFs from entering rooms. Another option is to get your house equipped with a conductive enclosure. These devices are called Faraday cages.
emf blocking of studies have proven that the non-ionizing RF EMF produces antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to result from the deregulation of cancer stem cells. This could explain the long-term responses seen in patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in patients with cancer is not evident.
Effects on the effects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumours were divided in three different groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effects of HCCMF on tumors was compared to that of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.
block emf behind tumour-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have wider implications for the diagnosis and treatment of various cancers.
The tumors in those in the group that were unaffected to EMF from RF, and fed a standard mouse diet. The tumors of the HCCMF group were infected with Huh7 cells when they were between five and seven weeks old. The tumours were then euthanized after they had a high burden.
block emf from the three groups also showed distinct growth curves. The HCCMF-treated tumors showed a significant decrease in tumour size after eight weeks. However, tumors treated with RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated by RCF were able to show necrosis that is common in tumours that are exposed to RCF. It is possible that the necrosis was due to the lack of oxygen in larger cancers.
In conclusion, the findings suggest the fact that AM EMF is a powerful source of anticancer effects in vitro and in the vivo. A number of studies have proven that AM RF EMF produces measurable shrinkage of tumors in HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the growth of HCC tumours in the vivo.