Ts Factor 10

Ts Factor 10

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Affiliation

1 Department of Veterans Affairs, Nashville, Tennessee, USA.

T S Blackwell et al.

Am J Respir Cell Mol Biol .

1997 Jul .

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1 Department of Veterans Affairs, Nashville, Tennessee, USA.

Blackwell TS, Blackwell TR, Holden EP, Christman BW, Christman JW.
Blackwell TS, et al.
J Immunol. 1996 Aug 15;157(4):1630-7.
J Immunol. 1996.

PMID: 8759749

Martin T, Cardarelli PM, Parry GC, Felts KA, Cobb RR.
Martin T, et al.
Eur J Immunol. 1997 May;27(5):1091-7. doi: 10.1002/eji.1830270508.
Eur J Immunol. 1997.

PMID: 9174597

Meduri GU, Muthiah MP, Carratu P, Eltorky M, Chrousos GP.
Meduri GU, et al.
Neuroimmunomodulation. 2005;12(6):321-38. doi: 10.1159/000091126.
Neuroimmunomodulation. 2005.

PMID: 16557033

Clinical Trial.

Sahnoun Z, Jamoussi K, Zeghal KM.
Sahnoun Z, et al.
Therapie. 1998 Jul-Aug;53(4):315-39.
Therapie. 1998.

PMID: 9806002

Review.
French.

Legrand-Poels S, Schoonbroodt S, Matroule JY, Piette J.
Legrand-Poels S, et al.
J Photochem Photobiol B. 1998 Aug 21;45(1):1-8. doi: 10.1016/S1011-1344(98)00118-3.
J Photochem Photobiol B. 1998.

PMID: 9819895

Review.

Lee JH, You HJ, Lee TY, Kang HJ.
Lee JH, et al.
Int J Mol Sci. 2022 May 7;23(9):5234. doi: 10.3390/ijms23095234.
Int J Mol Sci. 2022.

PMID: 35563624
Free PMC article.

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Mazumdar V, Joshi K, Nandi BR, Namani S, Gupta VK, Radhakrishnan G.
Mazumdar V, et al.
Infect Immun. 2022 May 19;90(5):e0006022. doi: 10.1128/iai.00060-22. Epub 2022 Apr 14.
Infect Immun. 2022.

PMID: 35420446

Chen KY, Chang CY, Hsu HJ, Shih HJ, Huang IT, Patel HH, Huang CJ.
Chen KY, et al.
Pharmaceuticals (Basel). 2022 Feb 24;15(3):287. doi: 10.3390/ph15030287.
Pharmaceuticals (Basel). 2022.

PMID: 35337084
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Sarkar A, Chamucero-Millares JA, Rojas M.
Sarkar A, et al.
Am J Respir Cell Mol Biol. 2022 May;66(5):479-480. doi: 10.1165/rcmb.2022-0028ED.
Am J Respir Cell Mol Biol. 2022.

PMID: 35238734
Free PMC article.

No abstract available.

Niemiec SM, Hilton SA, Wallbank A, Louiselle AE, Elajaili H, Hu J, Singh S, Seal S, Nozik E, Smith B, Zgheib C, Liechty KW.
Niemiec SM, et al.
Nanomedicine. 2022 Feb;40:102498. doi: 10.1016/j.nano.2021.102498. Epub 2021 Nov 26.
Nanomedicine. 2022.

PMID: 34838994
Free PMC article.

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Transcription factors are DNA-binding proteins that regulate gene expression. Nuclear factor-kappa B (NF-kappa B) is a critical transcription factor for maximal expression of many cytokines that are involved in the pathogenesis of inflammatory diseases, such as adult respiratory distress syndrome (ARDS) and sepsis syndrome. Activation and regulation of NF-kappa B are tightly controlled by a group of inhibitory proteins (I kappa B) that sequester NF-kappa B in the cytoplasm of immune/inflammatory effector cells. NF-kappa B activation involves signaled phosphorylation, ubiquitination, and proteolysis of I kappa B. Liberated NF-kappa B migrates to the nucleus, where it binds to specific promoter sites and activates gene transcription. The activation of NF-kappa B initiates both extracellular and intracellular regulatory events that result in autoregulation of the inflammatory cascade through modulation of NF-kappa B activation. Recently, activation of NF-kappa B has been linked to ARDS and has been shown to be a critical proximal step in the initiation of neutrophilic inflammation in animal models. Activation of NF-kappa B can be inhibited in vivo by treatment with antioxidants, corticosteroids, and the induction of endotoxin tolerance. Identification of more specific and efficacious inhibitors of NF-kappa B activation might prove beneficial for the treatment of cytokine-mediated inflammatory diseases.

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