Relaxation et orgasmes

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Relaxation et orgasmes



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Oui, il existe 10 types d’orgasmes différents. Voici comment avoir chacun d’entre eux

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L’orgasme féminin a été ces derniers temps l’objet de plusieurs études. Ce plaisir sexuel est recherché par toutes les femmes, qu’elles soient hétérosexuelles, lesbiennes ou bisexuelles. Mais saviez-vous qu’il existe non pas 1 orgasme, mais 10 types différents d’orgasme féminin ?
L’orgasme, qu’est-ce que c’est ?
Selon la gynécologue Sherry Ross, l’orgasme est un réflexe physique qui se produit lorsque les muscles se contractent quand l’excitation sexuelle atteint son apogée, puis se détendent. Chaque point culminant peut sembler différent en termes d’intensité et de durée, selon la manière et la partie du corps qui est excitée. En plus d’offrir une excitation physique, c’est aussi une excitation émotionnelle qui permet de se sentir plus proche du partenaire ou simplement d’être plus détendu. Certains types d’orgasme se concentrent sur le vagin uniquement tandis que d’autres surviennent lorsque d’autres zones dites érogènes sont excitées. Voici les différents types d’orgasme féminin. 
Le point G se trouve à mi-chemin entre l’ouverture vaginale et le col de l’utérus. Selon le Dr Ross, pour stimuler le point G vous devez le presser doucement et le caresser légèrement surtout lorsque votre conjointe est excitée. Le point G remplit de sang se gonflera et déclenchera une sorte de plaisir profond et qui va inonder le corps en entier.
Un orgasme mixte est une excitation qui se produit lorsque plus d’une zone érogène est stimulée en même temps. La pénétration vaginale avec une stimulation du point G et la stimulation clitoridienne est une combinaison qui permet d’expérimenter l’orgasme mixte. 
Même si l’orgasme clitoridien peut être le plus accessible et le plus facile, une mauvaise stimulation du clitoris peut le rendre difficile à atteindre. D’où l’importance de bien savoir exciter ce petit organe interne. Quand il est suffisamment excité, le clitoris se gorge de sang et sa taille augmente permettant ainsi une explosion de sensations toutes aussi agréables qu’intenses.
Le clitoris et le point G ne sont pas les seules zones de plaisir. Les experts disent qu’il y a des zones érogènes supplémentaires à l’intérieur du vagin qui, lorsqu’elles sont touchées de la bonne façon, peuvent mener à ce que l’on appelle un orgasme vaginal. Les zones à l’intérieur du vagin contiennent des nerfs qui semblent très sensibles pour beaucoup de femmes. 
Les hommes ne sont pas les seuls à éjaculer. Eh oui, certaines femmes sont aussi capables d’éjaculer lorsqu’elles atteignent le nirvana. D’après le Dr Ross, lorsque les femmes sont excitées ou stimulées sexuellement, il y a parfois expulsion de liquide des glandes autour de l’urètre ou de la surface antérieure du vagin. Le liquide est généralement clair et ne ressemble pas à de l’urine, et il peut y avoir une quantité modérée ou une grande quantité.
L’orgasme cervical est lorsque le cervix (col de l’utérus) est stimulé. Il se produit lorsque la femme est très excitée. Il est préférable de miser sur beaucoup de préliminaires si vous souhaitez atteindre l’orgasme cervical afin de rendre le col de l’utérus plus réceptif au toucher. Durant l’acte sexuel, vous pouvez passer de la position du missionnaire à la position de la levrette pour atteindre ce type d’orgasme.
Les seins et les mamelons en particulier sont des zones érogènes majeures. Ces derniers réagissent surtout lorsqu’on les touche et les caresse, parce qu’ils sont chargés de terminaisons nerveuses et leur peau est super sensible. Certaines femmes peuvent vraiment atteindre l’orgasme rien qu’en se faisant caresser et embrasser les mamelons. 
Le coregasme ou l’orgasme suite à l’exercice physique
Une étude de l’Université de l’Indiana a révélé que plusieurs femmes avaient connu l’orgasme ou le plaisir sexuel pendant qu’elles faisaient de l’exercice physique, notamment lorsque ces exercices étaient accompagnés d’un ballon entre les jambes, en faisant du vélo ou encore de l’équitation.
L’orgasme nocturne commence habituellement par un rêve érotique, ce qui entraîne une augmentation du flux sanguin vers les parties génitales ainsi qu’une relaxation majeure, ce qui permet au corps d’atteindre l’orgasme alors qu’une femme n’est même pas éveillée.
Contrairement aux hommes, les femmes n’ont pas de période réfractaire après l’orgasme. Selon le Dr Hall, elles peuvent atteindre un orgasme, et faire ensuite l’expérience de plusieurs autres orgasmes avec de nouvelles stimulations. Pour atteindre plusieurs autres orgasmes, il est recommandé de continuer à contracter les muscles pelviens (en les serrant et en les relâchant comme on fait pour retenir l’urine). Cela permet de maintenir un flux sanguin élevé, ce qui augmente la sensibilité et rend l’orgasme suivant plus facile à atteindre.
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Current medical literature does not describe precisely the activation and mechanisms of prostate orgasms. This brief review describes what we know about the anatomy and physiology of the prostate and its involvement in reproduction and especially its stimulation for sexual recreation. It is illustrated with a highly relevant case history. Clin. Anat. 31:81–85, 2018. © 2017 Wiley Periodicals, Inc.
Cancer of the prostate is the most commonly diagnosed cancer in Western world males and is the second cause of cancer deaths in men (Siegel et al., 2015 ). The male human prostate is a musculoglandular organ the size of a walnut; it surrounds the neck of the bladder and urethra and is itself surrounded by a complex of fascial structures. Ayala et al. ( 1989 ) studied histologically whole organ sections of 50 prostate glands and concluded that a ‘capsule’ of the prostate does not exist, it being a concentrically placed fibromuscular band surrounding the prostate that is an inseparable component of the prostatic stroma. Raychaudhuri and Cahill ( 2008 ) in an extensive literature search about the pelvic fascia that included periprostatic structures confirmed this conclusion. However, Ali et al. ( 2004 ) still used the term ‘prostatic capsule’ when describing nerves passing through it into the prostate body. The gland can be divided into three zones, namely—a peripheral zone (75% of the gland, the portion that surrounds the distal urethra), a central zone (5–8% of the gland, the zone that surrounds the ejaculatory ducts), and a transitional zone (20% of the gland that surrounds the proximal urethra) all enclosed by the fibromuscular band.
The complexity of the development of the prostate in humans from the embryonic urogenital sinus has been reviewed by Marker et al. ( 2003 ) and Santos and Taboga ( 2006 ). This embryonic structure is indistinguishable between male and female embryos until weeks 10–12 of gestation. It then differentiates under the influence of the androgens secreted initially by the fetal testes that maintain its embryonic and neonatal growth through activation of local paracrine factors (Thomson, 2001 ) finally creating male prostatic glandular activity at puberty (Isaacs, 1994 ).
The functions of the prostate are its involvement in the reproductive aspects of the male ejaculate (its procreative function) and its involvement in the ecstacy of the orgasm (its recreative function). While there are many studies of its reproductive function(s) there are relatively few that characterize its recreative functioning. Most of the information about this pleasurable function comes from anecdotal sources. There is a website dedicated to its recreative function (see section below on prostate and the internet).
It should be mentioned that there are suggestions that the gland also has a possible endocrine function. Kacker et al. ( 2014 ) reviewed the evidence that the gland contributed to the local and systemic concentrations of 5α-dihydroxytestosterone, a more potent androgen than its precursor testosterone from which it is converted by the prostate expressed isoenzyme 5α-reductase-2 (Kang et al., 2014 ). The concentration of the androgen in the prostate is 5–10 times greater than that of testosterone (Hay and Wass, 2009 ). The importance of the prostate function for male fertility is exemplified by males who have a deficiency of this isoenzyme (5α-reductase type 2 deficiency) as they have decreased sperm counts, low semen volume and failure of the semen to liquefy because of a deficiency of the prostate-specific antigen (a serine protease) that de-coagulates the gelled ejaculated semen.
The gland is abundantly innervated by the parasympathetic (hypogastric and pelvic nerves) and by the sympathetic (hypogastric ganglion). According to Gupta and McVary ( 2017 ) ‘there is widespread agreement that expulsion of the contents of the gland during emission is under adrenergic control while cholinergic nerves are secretomotor’. In regard to the sensory innervation of the prostate, McVary et al. ( 1998 ) state that the majority of the afferents to the ventral prostate is localised to sensory nerves from the L5 and L 6 segments of the spinal cord but there is a smaller degree of innervation from T13 to L2.
A collection of nerves that is located in the fascia covering the prostate is named the prostatic plexus. They arise from the lower (inferior) portion of the pelvic plexus and are distributed not only to the prostate but also to the corpora cavernosa of the penis and urethra. Injury or damage to these nerves impacts on the mechanisms of erection and can thus cause erectile dysfunction (see section below on orgasm and radical prostatectomy).
Dense neuropeptide Y innervation is present throughout the prostatic stroma but most studies have not found the neuropeptide to be involved in the contraction of the prostate (White et al., 2013 ).
The serial order of the male ejaculate is composed of secretions from the glands of Littré (lining the penile urethra), Cowper's (bulbourethral) gland, testicular, and epididymal fluid containing the spermatozoa, the prostate and finally the seminal vesicles (Levin, 2005a ). In this glandular company the prostate manufactures a highly complex secretion which becomes approximately 30% of the fluid volume of the ejaculate. It contains a large variety of constituents many having proposed or unknown function(s). These include citric acid (20–150 mM, function unknown), zinc [590 ± 45 SE µg/mL; Zaichick et al. ( 1996 ), possibly antibacterial], the enzyme prostatic specific antigen (PSA) that liquefies the coagulated semen after ejaculation, prostatic acid phosphatase, phosphorylcholine (specific substrate for previous enzyme), aminopeptidase, ATPase, spermine (Pegg, 2014 ) and spermidine (possible antibacterial activity, enzymatic breakdown gives semen its characteristic odor), prostasomes [small vesicles containing cholesterol, sphingomyelin, calcium, enzymes, and some 139 proteins—(Levin, 2005 b)], lipids (Scott, 1945 ) and phospholipids of which sphingomyelin constitutes about half the latter with phosphatidyl serine and ethanolamine plasmalogen most of the remainder (their functions are unknown). Semen also contains some 60 plus peptides and proteins (Tsai et al., 1984 ; Fung et al., 2004 ).
According to Baker and Bellis ( 1995 ) the prostate secretion provides the sperm with some protection from the seminal vesicle secretion that has spermicidal properties (Linderholmer, 1973 ).
The involvement of the prostate in the mechanisms of ejaculation was first promoted by Marberger ( 1974 ). He proposed that the distention of the prostatic urethra by the volume of the entering semen was the trigger for the initiation of the ejaculatory reflex and the theory was called ‘the prostatic pressure chamber trigger concept’. This speculative explanation was repeated by a number of authors (Jannini et al., 2002 ). Levin ( 2005a ) reviewed the evidence for this mechanism and found that there were important experimental studies with results against the concept, namely that there are ‘definite occasions where the ejaculatory mechanisms is activated yet no seminal fluids enter the prostatic urethra’. Giuliano and Clement ( 2005 ), in their review of ejaculation, agreed that ‘the expulsion phase of ejaculation can occur in the absence of urethral stimulation and that the prostatic pressure chamber concept does not definitively identify the ejaculation trigger’.
Unlike the female orgasm, where a number of competing descriptions for induced orgasms exist (see Levin, 2015 ), those for the male are limited (Zilbergeld, 1979 ; Otto, 1999 ). Surprisingly, neither Masters and Johnson ( 1966 ), Zilbergeld ( 1979 ), Margolis ( 2004 ), or Bancroft ( 2009 ) in their books on human sexual arousal mention those obtained from prostate stimulation in the male while even in the book on orgasm by Komisaruk et al. ( 2006 ) has only a single, very short paragraph of but two sentences.
The classic penile-induced male orgasm description is that of Masters and Johnson ( 1966 ) who characterized it into two separate stages. The first stage is initiated by the contractions of the various accessory organs beginning with the vasa efferentia of the testis, then the epididymis following through to the vas deferens with the contractions of the seminal vesicles. The prostatic contractions then occur. This stage is controlled by the thoracolumbar (T11-L2) neural pathway (Giuliano and Clement, 2005 ). In this stage the male has a feeling of ‘ejaculatory inevitability’ and the knowledge that ejaculation is coming and cannot be delayed. The second stage is the seminal fluid flowing into the distended urethral bulb and the penile urethra. The perineal musculature (mainly the bulbocavernosus muscle, Levin, 2005 ) then propels the semen along the penile urethra to be expelled forcibly in spurts from the penile meatus, this is mediated by the sacral (S2-S4) pathway (Giuliano and Clement, 2005 ). With each spurt a feeling of intense pleasure is generated which gradually subsides as the spurts decrease. Often nonverbal vocalizations occur with each spurt (Levin, 2006 ). If the pelvic muscles do not contract the semen emission is one of dribbling, powered by the peristaltic contractions of the urethra alone with little ecstatic pleasure (Newman et al., 1982 ). Although orgasm normally takes place concomitantly with ejaculation, the two processes are actually independent (Levin, 2003 ).While the prostate is involved in forming part of the ejaculate (as detailed above) it is also involved in ejaculation per se as its fibromuscular covering containing smooth muscle contracts clonically under its adrenergic innervation propelling the semen from the prostatic urethra into the penile urethra (White et al., 2013 ).
Published descriptions of the prostate-induced orgasm in academic and clinical literature have been thin on the ground (Levin, 2004 ). In an early article by Perry ( 1988 ) he described prostatic-induced orgasms as ‘emission type reflexive orgasms’ with occasional oozing of semen from the penis. Such a description applies to ejaculations that occur when the pelvic striated muscles (especially the bulbocavernous) are nonfunctional (Newman et al., 1982 ). A paradox to note is that when induced ejaculations are without pelvic contractions they are of poor erotic value as previously described yet intense erotic pleasure appears to be activated by prostate stimulation even when there are no pelvic contractions to create semen ejaculation.
Men can experience changes in their sexual responses after radical prostatectomy, the gold-standard treatment for localized cancer of the prostate. Early operations caused damage to the nerves that passed along the organ that subserved erection but later nerve sparing operations were designed to preserve this innervation. Koeman et al. (1994), for example, reported that in their series of prostatectomies ( n = 20) no patient could maintain a rigid erection but 5 could manage to have coitus with their partial erection. None experienced the sensation of ‘ejaculatory inevitability’. A few ( n = 7) complained that their orgasm was weakened and 9 had involuntary loss of urine at orgasm (climacturia). A very extensive, comprehensive, and up-to-date review of orgasmic dysfunctions after radical prostatectomy by Capogrosso et al. ( 2017 ) report that despite technical surgical advances ‘the achievement of good operative functional outcomes is still considered a troublesome issue both for patients and urologist’. These include impairments in sexual desire, penile morphology and orgasmic function. In relation to the latter the conditions of climacturia, orgasm associated pain and modification of orgasmic sensation are prevalent and even complete anorgasmia occurs. Unfortunately, reliable data from which to estimate these impairment risks are lacking.
An obvious question is—why do prostate orgasms appear more powerful and pleasurable than penile induced ones? Increased body awareness has been linked to increased genital awareness and arousal in women (see Handy and Meston, 2016 for references). It is possible that similar heightened awareness occurs in those males who focus on and practice prostate stimulation. Such awareness could enhance the sexual pleasure obtained as modulation of physiological function can occur through changes in mental processes (Mitani et al., 2006 ). It is now accepted that the human brain is constantly changing its functional and structural properties depending on the variety of inputs and experiences. This plasticity is thought
De l'échangisme pour la première fois
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