Older Penis In Sperm

Older Penis In Sperm
by Jay Ferrari and Marty Munson Published: Jul 27, 2020
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This is not a story about what might happen to your penis. It's not about weird accidents (sorry?), or what happens when you try a less-than-great way to try to make your penis bigger (although we do have that ).
These are things that will likely eventually happen to your penis, if they haven't already. Like other parts of your body, your penis is at the mercy of time as you age. Changes do occur. The penis you know now will not be the penis you will know 10 or 20 years from now.
(Pause provided to allow that fact to sink in.)
That's not to say that what happens to your penis as you age is bad thing. And that's also not to say that what happens to your penis as you age is inevitable.
That's the hopeful part: that you can fight against fate. Some of the things that follow you can prevent or delay, to a certain extent, and you don't need any weirdo products or convoluted exercises to achieve your anti-aging penis goals.
All you need is the help of Brian Steixner, M.D., the director of the Institute of Men’s Health for the Jersey Urology Group. We asked him for the top aging-penis-related scenarios he sees—and then how to best treat them.
The good news: You can change a couple things right now that could keep you from getting old man penis (if that's a worry you have).
According to Dr. Steixner—and verified by the naked guys over 70 shuffling around every health club locker room coast to coast—your scrotum will droop as you get older.
It’s a largely unavoidable aspect of aging thanks to a loss in muscle mass. At its most extreme, you develop what Steixner calls “splash down” syndrome, which is exactly what it sounds like: When you sit to use the toilet, your scrotum actually hits the water.
You can keep things where you want them with an increasingly popular procedure called scrotoplasty. And it's worth taking some action now: to prevent saggy balls , not smoking and staying at a healthy body weight can help as well.
No, not the out-of-the-pool constriction we all know. There is a progressive loss of size as you age. (But it's not like you develop a micropenis or anything.)
What does that equation look like? Normal skin cells, once hale and hearty, get replaced by non-elastic fibers, which just sort of reel in the whole apparatus.
Want to make it worse? Keep building up that beer gut. As your belly gets bigger, the fat pad pushes out and a larger penile percentage gets buried under the skin. Gain weight, loose length. Simple math.
“For every 30 pounds you shed, you add an effective half-inch in length,” says Dr. Steixner. Discover the best ways for men over 50 to lose weight. And it's probably worth trying the peak-performance for your penis diet now, too.
Into your autumn years, you develop curvature. You’ll dogleg left. You’ll pull right. Thanks to repeated trauma from seemingly harmless things like sports and sexual activity, scar tissue can accumulate along the length of your penis.
And if that isn’t unsettling enough, this scar tissue does not build up symmetrically. Where you were once an arrow, you become a bow.
“Into your 60s and 70s,” says Dr. Steixner, “it can get worse. I’ve seen patients (whose penises) look for all appearances like a question mark.”
Mercifully, there’s help for punctuation mark syndrome. Today, injectable medicines help release the accumulated plaques anchoring the scar tissue holding your penis in that curve.
It’s actually a botulinum toxin that does the trick. That’s right, Botox injections. Just bite your lip and think happy thoughts.
There are more than 30 million men with erectile dysfunction, and the reason behind it for older men boils down to blood loss. (Right now, numerous habits may be killing your erection .)
“Having ED is like having a heart attack of the penis,” explains Dr. Steixner. “And preventing it involves pretty much the same advice you give to someone with a heart condition. Eat well, exercise—control those, and you should be fine.”
Contending with ED is a billion-dollar market, with treatments from prescription pills and injections to boost blood flow. Recently, researchers have been developing therapies from topical gels to shock-wave devices (read about the latest developments here ), to treat this frustrating problem.
“If you take care of yourself, however, you can avoid all that,” Dr. Steixner says. “I have patients in their 90s who have perfect erections because they’ve taken care of themselves.”
Most men are familiar with the very real risks of prostate and testicular cancer, and symptoms of these are something to watch for as you age. But there’s also a risk of skin cancer on the penis itself, especially if you like to frequent tanning beds au natural.
Also, men who are uncircumcised but remiss in their personal hygiene can create conditions that contribute to penile cancer .
“Stay out of tanning beds, and if you are uncircumcised, keep the hood clean,” urges Dr. Steixner.
What’s the worst that can happen? Don’t ask.
Okay, you asked: “In extreme cases, a complete removal of the penis is required,” Steixner says. “It happens. Maybe once a year. I have a patient—"
Stop there, doc. Please. Just stop there. No tanning beds. Stay clean. We got it.
As for the less horrifying, more likely changes we talked about at first, know that as you age, your testosterone levels will decrease. That means slight but consistent loss in size. Decreased nerve function and sensitivity may give you some difficulty achieving orgasm.
In other words, your old man penis is going to last longer, but get shorter. Just don’t forget that it could be worse. So, so much worse.
8 Signs You May Have Erectile Dysfunction
Everything You Want to Know About Penis Rings
Where to Get Nitroglycerin Gel for ED
25 Foods That Might Help You Stay Erect
5 Harmless Reasons You Can't Get An Erection
Why Your Deodorant Might Be Making You Flabby, Tired, and Impotent
6 Ways Your Diet Is Destroying Your Sex Life
11 Ways to Deal With Erectile Dysfunction
10 Myths About Erectile Dysfunction You Must Stop Believing
Happy Holidays: Viagra is About to Get Way Cheaper
You Should Never Buy Viagra Without a Prescription. Here's Why
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6 To whom correspondence should be addressed at: ANZAC Research Institute, Sydney, NSW 2139, Australia. Email: djh@anzac.edu.au

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Human Reproduction , Volume 19, Issue 8, 1 August 2004, Pages 1811–1815, https://doi.org/10.1093/humrep/deh315

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K.K. Ng, R. Donat, L. Chan, A. Lalak, I. Di Pierro, D.J. Handelsman, Sperm output of older men, Human Reproduction , Volume 19, Issue 8, 1 August 2004, Pages 1811–1815, https://doi.org/10.1093/humrep/deh315
BACKGROUND: Declining fertility of couples from the fourth decade of life is largely attributable to the drop in female fertility. However, increasing numbers of men, whose fertility theoretically lasts until death, are seeking fertility treatment at older ages, yet there is little information on sperm production and function past the age of 50 years. The few studies of such older men have examined men attending fertility clinics, and therefore willing to provide semen samples, but the participation bias of such recruitment hinders extrapolation to the unselected general male population. METHODS: We have taken the opportunity to study a convenience sample of 55 healthy, non-infertile men ranging in age from 52 to 79 years old who provided semen samples as part of a prostate cancer screening project. They were compared with a control group ( n =409) of younger (<52 years) men from among 567 volunteers screened as potential sperm donors for an artificial insemination program. RESULTS: Older men had lower semen volume (mean semen volume 1.8 versus 3.2 ml; P <0.0001) and total sperm output (median 74 versus 206 million sperm per ejaculate; P <0.0001), whereas sperm density (median 64 versus 73 million sperm/ml; P =0.12) was non-significantly decreased. Older men had more abnormal sperm morphology with decreasing numbers of normal forms (mean 14% versus 25%; P <0.0001) and reduced vitality (mean 51% versus 80%; P <0.0001), as well as increased numbers of cytoplasmic droplets (median 1 versus 0; P <0.0001) and sperm tail abnormalities (30% versus 17%; P <0.0001). Sperm head or neck abnormalities were no different between the groups. CONCLUSIONS: While neither study group may be representative of the general male population, these findings suggest that sperm production, reflected in sperm output but not sperm density, as well as sperm morphology and viability are diminished in this population of healthy, non-infertile older men.
It is well known that the fertility of couples declines with age ( Leridon, 1977 ; Schwartz and Mayaux, 1982 ; Menken et al. , 1986 ). This is mostly attributable to declining female fertility evident from the age of 30 years ( Schwartz and Mayaux, 1982 ) and ceasing altogether by menopause. This precipitate decline in female fertility, together with couples usually being very closely matched in age, overshadows and complicates efforts to determine whether there are also significant declines in male fertility with age ( Kidd et al. , 2001 ). Very few studies have addressed actual fertility of older men controlling for declining female fertility ( Anderson, 1975 ), although the time to pregnancy questionnaire ( Joffe, 2003 ) is promising ( Hassan and Killick, 2003 ), but has yet to be applied to older men.
Although testicular function does not exhibit a precipitous age-related decline like the ovary at menopause, there is a gradual and variable decline of modest proportion in testosterone production in older men ( Gray et al. , 1991b ; Harman et al. , 2001 ). Whether there is any real decline in spermatogenesis and/or male fertility in the general male population is much less clear. As direct sampling of the human testis is not feasible for population studies, sperm output is widely used as the surrogate measure of human male fertility. However, men are reluctant to provide semen samples unless actively concerned about their fertility. For example, population-based studies typically recruit <20% of young men willing to provide semen samples ( Jensen et al. , 2004 ) constituting an inevitable participation bias in such studies ( Handelsman, 1997 ; Cohn et al. , 2002 ). The limited number of published studies of sperm output in older men are largely restricted to men attending infertility clinics, where few are older than 50 years ( Kidd et al. , 2001 ). An uncertain, but probably high, proportion of such men have unrecognized defects in sperm production and/or function. Furthermore, access to such specialized medical services may be strongly influenced by non-biological factors, and findings from infertility clinics may not be reliably extrapolated to the general male population. Hence, few studies of older men have managed to avoid severe participation and selection biases.
Therefore, in order to provide novel insight into sperm output among healthy, non-infertile older men, we took the opportunity to study a convenience sample of healthy older men without known reproductive disorders or prostate disease who provided semen samples for prostate cytology as part of medical screening for undiagnosed prostate disease ( Gardiner et al. , 2003 ).
The sample of older men comprised 55 consecutive men who were referred by urologists from private office practices for a prostate cancer detection program based on seminal cytology ( Gardiner et al. , 1996 ; Clements et al. , 1999 ). All these men were asymptomatic and had been identified by elevated blood prostate-specific antigen (PSA) concentrations that required prostate biopsy. The men provided a single semen sample to the Clinical Andrology laboratory on the same day immediately prior to their transrectal ultrasound and prostate biopsy for possible in situ prostate cancer. The diagnostic outcomes of the prostate cytology will be reported separately.
The control group comprised 409 me
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