Mom1
Mom1 MOM1. Organism. Arabidopsis thaliana (Mouse-ear cress). Status. Reviewed-Annotation score.
impaired gene silencing with normal methylation. CS · mom · CS images. None available. phenotypes .
MOM1 is part of a silencing pathway that acts independently of DNA methylation (Mittelsten Scheid et al, ). We assessed whether this.
At the whole genome level, MOM1 regulates transcriptional silencing without alteration of DNA methylation [15,16]. Mutation in NRPE1, the.
This entry represents a group of plant proteins, including MOM1 from Arabidopsis. MOM1 is involved in transcriptional gene silencing (TGS).
Two Arabidopsis SUMO E3 ligase-like proteins interact with MOM1 to form a complex that mediates transcriptional silencing independently of changes in DNA.
The TGS regulator MOM1 in Arabidopsis is exceptional since it regulates transcription in intermediate heterochromatin with only minor changes in epigenetic.
The Mom1 (Modifier of Min-1) region of distal chromosome 4 was identified during a screen for polymorphic modifiers of intestinal.
(B) Northern blot showing levels of TSI transcripts of four wild-type plants (WT) and five plants of each mutant strain; pkl, mom1 and mom1pkl double mutant.
Quantitative trait loci studies have identified a locus, Mom1, which maps to the distal region of chromosome 4, that dramatically modifies Min-induced tumor.
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Symbol: Pla2g2aMom1-r. Name: phospholipase A2, group IIA (platelets, synovial fluid); modifier of Min1, tumor resistance. MGI ID: MGI
The heterochromatic regions around centromeres of animal and plant chromosomes are composed of tandem repetitive sequences, interspersed with transposons.
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For the Mom1 modifier of intestinal adenomas caused by ApcMin, these tests are used to evaluate two candidate genes: Pla2g2a, a secretory phospholipase, and.
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The secretory phospholipase Pla2g2a is a candidate for Mom1. Here, we investigate the range of action of Apc and Mom1. Analysis of chimeric Min mice indicates.
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We recently revealed that MORPHEUS' MOLECULE1 (MOM1) of Arabidopsis thaliana silences endogenous loci related to transposons and homologous to the nt.
Quantitative trait loci studies have identified the Mom1 locus (for modifier of Min-1), which is responsible for part of the genetic variability in polyp number.
mom1. By Jeremy Loudenback. About the Author. Jeremy Loudenback. Jeremy Loudenback is a senior reporter for The Imprint.
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Most of the endogenous loci silenced by MOM1 are remnants of transposons that are clustered around centromeres, and therefore, RNAs transcribed from such.
MOM1, a protein of 2, amino acids that acts as a transcriptional silencer, represents such an exception.
transgene in the mom1 mutant. These results suggest that cytosine methyla- tion is not sufficient to enforce TGS,. IN THIS ISSUE. Mum's the Word: MOM and.
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The AKR/J (AKR) strain carries alleles that correlate with a strong reduction in tumor multiplicity. To study the effect of one of these modifiers, Mom1, we.
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Tumor multiplicity in Min mice is influenced by genetic modifier loci. Previously, we mapped one of these modifier loci, Mom1, to distal mouse chromosome 4.
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The mechanism by which MORPHEUS' MOLECULE1 (MOM1) contributes to transcriptional gene silencing has remained elusive since the gene was first.
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Contact Cusabio to Customize MOM1 Antibody, which reacts with Arabidopsis thaliana (Mouse-ear cress) and will validated for ELISA, WB (ensure identification.
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Quantitative trait loci studies have identified a locus, Mom1, which maps to the distal region of chromosome 4, that dramatically modifies Min -induced tumor.
View dendrogram porcupine MDYQYFEEESDYIDLDEEEEDDDVVTAGSLDHRFGQPNGEEDYYFGGDDVEEELVVDGHG MOM1 MDNGHGYEEFELLEDGSLGECSTQVANATFLTISKLICLVVAFKFIKHIPISTTVRVYVH.
The MOM1 FISH probe is designed to hybridize to the MOM1 gene and is primarily used for detecting amplifications and deletions associated with the gene.
Chez Arabidopsis, la proteine MOM1 (Morpheus' Molecule 1) est impliquee dans le silencing epigenetique des genes. La mutante "mom1" ne presente aucun.
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