Галерея 3365602

Галерея 3365602




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Галерея 3365602

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Affiliation



1 Department of Physiology, Faculty of Medicine, Kuwait University, Safat, Arabian Gulf.







R I Jabr et al.






Can J Cardiol .



1988 Mar .







Format


Abstract

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Affiliation



1 Department of Physiology, Faculty of Medicine, Kuwait University, Safat, Arabian Gulf.





Juggi JS, Braveny P, Telahoun G, Shuhaiber HJ, Yousof AM.
Juggi JS, et al.
Adv Myocardiol. 1985;6:491-502.
Adv Myocardiol. 1985.

PMID: 3992047








Smith PK, Buhrman WC, Ferguson TB Jr, Levett JM, Cox JL.
Smith PK, et al.
Circulation. 1983 Sep;68(3 Pt 2):II41-8.
Circulation. 1983.

PMID: 6872194








Lu K, Otani H, Yamamura T, Nakao Y, Hattori R, Ninomiya H, Osako M, Imamura H.
Lu K, et al.
J Thorac Cardiovasc Surg. 2001 Jan;121(1):137-48. doi: 10.1067/mtc.2001.111210.
J Thorac Cardiovasc Surg. 2001.

PMID: 11135170








Cinca Cuscullola J.
Cinca Cuscullola J.
Rev Esp Cardiol. 1988 Apr;41(4):244-53.
Rev Esp Cardiol. 1988.

PMID: 3045923


Review.
Spanish.
No abstract available.



Ip JH, Levin RI.
Ip JH, et al.
Am Heart J. 1988 May;115(5):1094-104. doi: 10.1016/0002-8703(88)90082-8.
Am Heart J. 1988.

PMID: 3284312


Review.

No abstract available.



Yi J, Duan H, Chen K, Wen C, Cao Y, Gao H.
Yi J, et al.
J Cardiovasc Transl Res. 2022 Dec;15(6):1464-1473. doi: 10.1007/s12265-022-10256-7. Epub 2022 Jun 10.
J Cardiovasc Transl Res. 2022.

PMID: 35689125







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Normothermic ischemic arrest of the perfused rat heart for 1 h was followed by severe reperfusion arrhythmias, depressed excitability, atrioventricular (AV) node and sinoatrial (SA) node conduction blocks and by marked impairment of contractility. Excitability and conduction recovered within 10 to 15 mins whereas contractility remained greatly depressed. This finding is in agreement with a theoretically based assumption of lesser sensitivity of conduction system to ischemia compared to the working myocardium. When the hearts were exposed to ischemia at 20 degrees C, the sinus rhythm recovered instantaneously upon reperfusion; excitability and SA node conduction times were not significantly different from control; and contractility was much less depressed than after 34 degrees C ischemia. However, AV node conduction times did not normalize even after 15 mins of reperfusion. Potassium cardioplegia (34 degrees C) did not prevent post ischemic arrhythmias but evidently protected both SA node and AV node conduction. Optimum recovery of the conduction system and contractility was achieved by hypothermic (20 degrees C) potassium cardioplegia. Other cardioplegic additives did not further improve the hypothermic potassium protection of the conduction system.


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