Галерея 3311620
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Галерея 3311620
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[offer god] 拜得神多自有神庇佑 offer god (45)
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【All-in-One and One-in-All】《病港II》(62)
不會輕易放棄汽油車! Toyota傳將於2024年推出新版Camry及RAV4
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Capcom網上發佈會「Capcom Spotlight」預定3月10日朝早6:30播出
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機械人產品綜合討論區 #28 謝絕老翻同炒家 歡迎互毒 新一年祝大家買多啲!
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鄭秀文 Sammi 出左新歌《愛是…2.0》!(ft. 好青年荼毒室〉
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[D369-] 抗俄衛國 153: 習大大 all in or not all in
點解唔同嘅派系
名字都同個輩分有關
華山 「風」 「不」
青城 「人」
恒山「定」 「儀」
點解偏偏令狐沖呢一代就唔跟規矩
無晒字號
Erstelle ein Konto, um deinen Lieblingscommunities zu folgen und mitzureden.
Gepostet von u/[deleted] vor 3 Jahren
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Well at least the dick makes up for the lack of nuts
Damn i was looking for something to crunch on...
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Affiliation
1 Laboratory of Analytical and Quantitative Cytology, Geneva, Switzerland.
E Johannisson .
Contraception .
1987 Jul .
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1 Laboratory of Analytical and Quantitative Cytology, Geneva, Switzerland.
Sheppard BL.
Sheppard BL.
Contraception. 1987 Jul;36(1):1-10. doi: 10.1016/0010-7824(87)90057-6.
Contraception. 1987.
PMID: 3117492
Review.
Sheppard BL, Bonnar J.
Sheppard BL, et al.
Contracept Fertil Sex (Paris). 1987 Nov;15(11):1015-24.
Contracept Fertil Sex (Paris). 1987.
PMID: 12341565
French.
Ortiz ME, Croxatto HB, Bardin CW.
Ortiz ME, et al.
Obstet Gynecol Surv. 1996 Dec;51(12 Suppl):S42-51. doi: 10.1097/00006254-199612000-00014.
Obstet Gynecol Surv. 1996.
PMID: 8972502
Review.
Sheppard BL, Bonnar J.
Sheppard BL, et al.
Am J Obstet Gynecol. 1983 Aug 1;146(7):829-39. doi: 10.1016/0002-9378(83)91087-6.
Am J Obstet Gynecol. 1983.
PMID: 6869454
Clark DA, Wang S, Rogers P, Vince G, Affandi B.
Clark DA, et al.
Hum Reprod. 1996 Jul;11(7):1438-44. doi: 10.1093/oxfordjournals.humrep.a019416.
Hum Reprod. 1996.
PMID: 8671483
Han L, Ma S, Zhao L, Liu Y, Wang Y, Feng X, Zhang K, Wang L, Wang L, Yin P, Liang D, Hou H, Shi G, Li Q.
Han L, et al.
Front Med (Lausanne). 2020 Nov 30;7:598689. doi: 10.3389/fmed.2020.598689. eCollection 2020.
Front Med (Lausanne). 2020.
PMID: 33330563
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Chua KJ, McLucas B.
Chua KJ, et al.
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Buskmiller C, Harrison D, Ruppersberger LA, Yeung PP Jr.
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PMID: 32431449
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Averbach S, et al.
Contraception. 2018 Apr 17:S0010-7824(18)30144-6. doi: 10.1016/j.contraception.2018.04.008. Online ahead of print.
Contraception. 2018.
PMID: 29673740
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Averbach SH, Ma Y, Smith-McCune K, Shiboski S, Moscicki AB.
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Am J Obstet Gynecol. 2017 Apr;216(4):386.e1-386.e5. doi: 10.1016/j.ajog.2016.11.1053. Epub 2016 Dec 14.
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The mechanism of action of intrauterine devices varies from one type of device to another, although the principle of action is the same, namely, to interfere with the physiology of reproduction at the endometrial level. The biochemical changes of the endometrial tissue and the composition of the uterine fluid have been reviewed in relation to the use of inert, copper-medicated and steroid-releasing IUDs. All IUDs, whether inert or medicated, provoke a significant increase in the number of neutrophils, mononuclear cells and plasma cells, the presence of which is unrelated to the tissue infiltration of inflammatory cells occurring pre-menstrually of the normal menstrual cycle. The increase of leucocytes in IUD-users is compatible with a foreign body reaction which may be related to the antifertility effect. The addition of copper to an inert IUD has been shown to significantly alter the metabolism of the endometrial cells, e.g. the enzymatic activity and the DOVA-synthesis. The steroid-medicated IUDs represent a new approach to intrauterine contraception where the morphology of the endometrium is considerably altered, showing massive decidual changes, atrophic glands and sometimes atrophy of the whole functional layer. In addition, there is an important foreign body reaction, similar to that of the inert IUDs. The enzymatic activity, as well as the proliferative activity was significantly altered in the endometrium of these IUD-users. Changes in the endometrial fibrinolytic activity in IUD-users have been demonstrated to be related to the presence of irregular bleeding. Micro-traumata of the endometrium and the increased fibrinolytic activity may also interfere with the protective processes present in the physiologically normal endometrium. Further studies on the factors influencing the fertilizing ability of the spermatozoa in the female genital tract, as well as on the conditions of the human endometrium required for the implantation of a blastocyst, may yield important information for the improvement of intrauterine contraception.
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