EMF Blocking plus EMF Shielding
No matter if you live in an apartment or house or simply would like to ensure your home is free of EMFs There are plenty of ways you can reduce exposure. One of the easiest is to limit your electronic device use. You could also consider EMF block paint to stop EMF radiation from reaching your house. Another way to shield your home from EMF radiation is to put up a RF shielding canopy. This is a cloth of net that contains EMF shielding and is used to block EMFs from entering a room. Another option is to have your home fitted with an electrical enclosure. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF has antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to involve down-regulation of cancer stem cells. This may account for the long-term responses seen in certain patients suffering from advanced HCC. However, the mechanism behind AM RF EMF's effect in cancer patients is not yet clear.
The effects on the effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were split in three different groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to the one used by humans. emf blockers were exposed the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.
The mechanism behind cancer-specific AM RF EMF might be due to the fact that tumor cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated through CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications in the treatment and diagnosis of many cancers.
The tumours in the control group were not exposed to RF EMF, and were fed a normal diet of mice. emf blocker of those in the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours from the three groups showed different growth curves. The tumours treated with HCCMF saw a significant reduction in tumour size after 8 weeks. However, tumors that were treated using RCF didn't show shrinkage. emf blockers was substantial. The tumors treated with RCF had necrosis that is common when tumors are that are exposed to RCF. The possibility is that this necrosis was caused by the lack of oxygen in larger tumours.
In sum, the results suggest an AM-RF EMF is a powerful source of anticancer effects in vitro and in live. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting effect on the growth of HCC tumours in living tissue.