EMF Blocking plus EMF Shielding
No matter if you live in a house or apartment or just would like to ensure your home is free of EMFs There are a variety of methods to reduce exposure. One of the easiest is to restrict your electronic device use. You could also consider EMF blocker paint to block EMF radiation from reaching your house. Another easy way to protect your home against EMF radiation would be to install a shielding canopy for RF. This is a cloth of net that contains EMF shielding and is used to prevent EMFs from entering a room. Another option is to have your house equipped with an enclosure that is conductive. emf neutralizer are referred to as Faraday cages.
Several studies have shown that the non-ionizing RF EMF produces antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation the cancer stem cell. This could explain the long-term effects observed in patients suffering from advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not evident.
The effects of AM RF EMFs on HCC tumor growth in vivo were examined in mice. The tumours were divided into 3 groups. The first group was not exposed to RF EMF. The second group was exposed to RF EMF at the same frequency to the frequency used by humans. The third group was exposed to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results indicated that tumours treated with HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is mediated through CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications for treatment and diagnosis of many cancers.
The tumours of those in the group that were unaffected EMF from radiofrequency, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumors were removed in cases of excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, tumors which were treated by RCF showed no signs of shrinkage. The difference was significant. The tumors treated with RCF had necrosis that is common in tumours exposed to RCF. It is possible that the necrosis is caused by the lack of oxygen in the more invasive cancers.
In sum, the results show that AM RF EMF is a powerful source of anticancer activity in vitro and in live. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors in HCC patients. The possibility is that the AM EMF causes these effects through CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. In emf neutralizer , AM RF EMF may exert a sustained effect on the development of HCC tumours in the vivo.