EMF Blocking plus EMF Shielding
No matter if you live in a house or apartment or simply want to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. It is also possible to use EMF block paint to stop EMF radiation from entering your house. Another easy way to protect your house from EMF radiation is to install an RF shielding canopy. This is a cloth made of net that has EMF shielding and is used to prevent EMFs from entering a space. Another option is to have your home fitted with a conductive enclosure. These enclosures are known as Faraday cages.
Several studies have shown studies have shown that nonionizing EMF can cause antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could be the reason for the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM EMF's effects on cancer patients is not yet clear.
Effects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumors were split into 3 groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at a frequency that is similar to that used in humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. emf blockers on HCC cells is controlled through CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H may have broader implications for diagnosis and treatment of various cancers.
The tumours in the control group were not exposed to EMF from RF, and fed a normal diet of mice. The tumors in HCCMF HCCMF group were infected with Huh7 cells at the time they were five-seven weeks old. emf blocking were then euthanized in cases of excessive burden.
The tumours in the three groups also displayed distinct growth curves. The tumours treated with HCCMF saw a significant reduction in tumour size after 8 weeks. However, the tumors which were treated by RCF showed no signs of shrinkage. The difference was significant. block emf treated with RCF showed necrosis, which is typical in tumors that have been exposed to RCF. There is a possibility that this necrosis is caused by a lack of oxygen in the larger tumours.
In summary, the results suggest an AM-RF EMF is a powerful source of anticancer activity in vitro and in the vivo. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF causes these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting impact on the growth of HCC tumors in vivo.