EMF Blocking in addition to EMF Shielding
If you reside in an apartment or house or just need to ensure that your house is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the most effective is to limit your use of electronic devices. You can also turn to EMF block paint to stop EMF radiation from reaching your home. Another method to protect your home from EMF radiations is to install a shielding canopy for RF. This is a cloth of net that contains EMF shielding and is used to stop EMFs from entering a room. Another option is to have your home fitted with a conductive enclosure. They are referred to as Faraday cages.
Several studies have shown that the non-ionizing RF EMF produces antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. This may account for the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients is not evident.
Aspects from AM RF EMFs on HCC tumour growth in vivo were studied in mice. The tumors were split into three groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the frequency used by humans. Third group members were exposed the RF EMF in HCC-specific frequencies. http://beatmimosa6.xtgem.com/__xt_blog/__xtblog_entry/__xtblog_entry/33690757-information-about-emf-blocking-radiation?__xtblog_block_id=1#xt_blog of HCCMF on the tumours was evaluated against that of RCF. The results indicated that cancers treated with HCCMF had significant shrinkage. However, tumours treated with RCF showed no evidence of shrinkage of the tumor.
The reason for tumour-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is controlled by CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications for the diagnosis and treatment of various cancers.
The tumors in the control group were not exposed to EMF from RF, and fed a normal diet of mice. The tumors of the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.
The tumours in the three groups also showed distinct growth curves. https://carlton-hampton.thoughtlanes.net/emf-block-paint-in-addition-to-emf-shielding-1681441404 treated with HCCMF had a significant reduction in the size of the tumour after 8 weeks. However, tumors that were treated using RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis, which is typical in tumours exposed to RCF. There is a possibility that this necrosis is caused by the lack of oxygen in the larger tumours.
In sum, the results show that AM RF EMF is a powerful source of anticancer activity in vitro and in live. Several studies have shown the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF triggers these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the growth of HCC tumors in the vivo.