EMF Blocking in addition to EMF Shielding
No matter if you live in an apartment or a house or just want to keep your home free from EMFs There are a variety of methods to reduce exposure. One of the most effective is to restrict your electronic device use. You can also turn to EMF block paint to prevent EMF radiation from reaching your house. emf blockers to protect your home from EMF radiations is to use a shielding canopy for RF. This is a fabric of net that contains EMF shielding. It is utilized to block EMFs from entering a space. Another option is to get your house equipped with a conductive enclosure. These enclosures are known as Faraday cages.
Several studies have shown that the non-ionizing RF EMF produces antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to result from the deregulation of cancer stem cells. This could be the reason for the long-term response seen in some patients with advanced HCC. But, the reason for AM RF EMF's effect in cancer patients is not evident.
Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were studied in mice. The tumors were split into three groups. First, the group that was unaffected RF EMF. Second group members were exposed to RF EMF at a frequency that is similar to that used in humans. In emf blockers , they were subjected the RF EMF in HCC-specific frequencies. block emf of HCCMF on the tumours was assessed against the effect of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is mediated through CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications in the treatment and diagnosis of many cancers.
The tumours in the control group were not exposed to EMF from RF, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were treated with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours from the three groups also showed distinct growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumors treated with RCF showed no shrinkage. The difference was substantial. The tumors treated by RCF showed necrosis, which is common when tumors are that are exposed to RCF. There is a possibility that this necrosis is caused by the lack of oxygen in the more invasive tumors.
In sum, the results indicate that AM RF EMF exhibits anticancer effects in vitro and in the vivo. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors for HCC patients. There is a possibility that AM RF EMF produces these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the growth of HCC tumours in living tissue.