EMF Blocking in addition to EMF Shielding
Whether you live in an apartment or a house or just need to ensure that your house is free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to restrict your use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another easy way to protect your house from EMF radiation is to put up a shielding canopy for RF. It is a type made of net which contains EMF shielding. It is utilized to stop EMFs from entering rooms. Another option is to have your house equipped with a conductive enclosure. These devices are called Faraday cages.
Several studies have shown how the non-ionizing energy of RF produces antiproliferative effects in HCC cells. emf blocking that drives AM RF EMF's anticancer activity in vitro is believed to involve down-regulation in cancer-related stem cells. This could explain the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients is not clear.
emf blockers on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumors were split into three groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at the same frequency to the one used by humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on the tumours was assessed against the effect of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF showed no evidence of tumour shrinkage.
The reason for tumour-specific AM RF EMF could be based on the fact that tumor cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is controlled by CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have more broader implications for diagnosis and treatment of various cancers.
The tumors in the control group were not exposed RF EMF, and were fed a normal diet of mice. The tumors in the HCCMF group were treated with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.
The tumours in the three groups also showed different growth curves. The tumours treated with HCCMF showed a significant decrease in the size of the tumour after 8 weeks. However, Find out more that were treated using RCF showed no reduction in size. The difference was significant. The tumors treated with RCF had necrosis that is common in tumors that have been that are exposed to RCF. It is possible that this necrosis was caused by the lack of oxygen in the more invasive tumors.
In summary, the results show the fact that AM EMF has anticancer activity in vitro and in vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. It is possible that AM RF EMF triggers these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting impact on the development of HCC tumours in the vivo.