EMF Blocking and even EMF Shielding
No matter if you live in an apartment or a house or simply need to ensure that your house is free of EMFs There are plenty of ways you can reduce exposure. One of the easiest is to limit your use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your house. Another method to protect your home against EMF radiation would be to use a shielding canopy for RF. This is a fabric of net that contains EMF shielding. It is utilized to block EMFs from entering a space. Another option is to get your home equipped with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven studies have shown that nonionizing EMF can cause antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This could explain the long-term response seen in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in patients with cancer is not yet clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were split into three groups. emf blocker did not have exposure RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the one used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on the tumours was assessed against the effect of RCF. The results showed that the cancers treated with HCCMF were significantly shrinking. However, the tumours treated with RCF did not show any evidence of tumour shrinkage.
emf blocking of tumour-specific AM RF EMF could be due to the fact that tumor cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is controlled through CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have more broader implications for diagnosis and treatment of various cancers.
The tumours in those in the group that were unaffected EMF from RF, and fed a normal diet of mice. The tumours in those in the HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed distinct growth curves. The HCCMF-treated tumors saw a significant reduction in size of the tumor after 8 weeks. However, tumors that were treated using RCF didn't show reduction in size. The difference was substantial. emf blockers treated with RCF were able to show necrosis that is common in tumors that have been exposed to RCF. There is a possibility that this necrosis was caused by the lack of oxygen in the larger cancers.
In conclusion, the findings show that AM RF EMF exhibits anticancer activity in vitro and in vivo. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF causes these effects because of CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting effect on the growth of HCC tumours in living tissue.