EMF Blocking and even EMF Shielding
If you reside in an apartment or house or simply would like to ensure your home is free from EMFs There are plenty of methods to reduce exposure. https://lake-justesen.mdwrite.net/details-of-emf-blocking-radiation-1681268857 of the easiest is to reduce the use of electronic devices. emf blocking is also possible to use EMF block paint to stop EMF radiation from reaching your home. Another easy way to protect your house from EMF radiations is to use an RF shielding canopy. This is a cloth made of net which contains EMF shielding and is used to stop EMFs from entering a room. Another alternative is to have your home equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
A number of studies have proven studies have shown that nonionizing EMF can cause antiproliferative properties in HCC cells. emf blockers of AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism behind AM RF EMF's effect in cancer patients is not clear.
Aspects of AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. The first group was not exposed RF EMF. The second group was exposed to RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumours was assessed against the effect of RCF. The results showed that the tumors treated by HCCMF had significant shrinkage. However, tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is controlled through CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have more broader implications in the diagnosis and treatment of various cancers.
The tumors in those in the group that were unaffected EMF from radiofrequency, and fed a normal mouse diet. The tumors in those in the HCCMF group were injected with Huh7 cells at the time they were five-seven weeks old. The tumors were removed after they had a high burden.
The tumours from the three groups also displayed different growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after eight weeks. However, the tumors which were treated by RCF didn't show reduction in size. The difference was significant. The tumours treated with RCF showed necrosis that is common in tumors that have been exposed to RCF. It is possible that the necrosis was due to a lack of oxygen in the larger tumors.
In summary, the results show an AM-RF EMF is a powerful source of anticancer activity in vitro and in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable reduction in tumours for HCC patients. The possibility is that the AM EMF causes these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the development of HCC tumours in living tissue.