EMF Blocking and even EMF Shielding
If you reside in an apartment or a house or just need to ensure that your house is free of EMFs, there are a number of methods to reduce exposure. One of the easiest is to restrict your electronic device use. block emf is also possible to use EMF block paint to prevent EMF radiation from entering your house. Another method to protect your house from EMF radiation would be to install a shielding canopy for RF. It is a type made of net which contains EMF shielding. It's used to block EMFs from entering rooms. Another alternative is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. This could explain the long-term response seen in certain patients suffering from advanced HCC. However, the mechanism behind AM RF EMF's effect in patients suffering from cancer isn't clear.
Aspects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were examined in mice. The tumours were divided into three groups. First, emf blocker that was unaffected RF EMF. The second group was exposed to RF EMF at a frequency that is similar to that used in humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumors was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is caused through CACNA1H which is a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have more broader implications in the treatment and diagnosis of different cancers.
block emf in the controls were never exposed EMF from RF, and fed a normal mouse diet. The tumors in the HCCMF group were infected with Huh7 cells at the time they were five-seven weeks old. The tumors were removed when they showed excessive burden.
The tumours in the three groups showed different growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, the tumours which were treated by RCF showed no shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis that is common in tumours that are exposed to RCF. There is a possibility that this necrosis was caused by an absence of oxygen in the more invasive cancers.
In conclusion, the findings indicate an AM-RF EMF exhibits anticancer effects in vitro and in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable shrinkage of tumors for HCC patients. It is possible that AM RF EMF produces these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting effect on the development of HCC tumors in the vivo.