EMF Blocking and even EMF Shielding
If you reside in a house or apartment, or you just want to keep your home free of electromagnetic fields, there are a number options to limit your exposure. One of the easiest is to limit your use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your home. Another easy way to protect your home from EMF radiation would be to use a RF shielding canopy. It is a type made of net that has EMF shielding. It's used to block EMFs from entering rooms. Another option is to have your home equipped with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF can cause antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to result from the deregulation the cancer stem cell. This could explain the long-term responses seen in patients suffering from advanced HCC. But, the reason for AM EMF's effects on patients suffering from cancer isn't evident.
Aspects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. https://note1s.com/notes/KZ8T4ZV were split into three groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at the same frequency to the frequency used by humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumors treated by HCCMF had significant shrinkage. However, emf neutralizer treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism behind tumor-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is controlled by CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications for treatment and diagnosis of different cancers.
The tumours in the controls were never exposed RF EMF, and were fed a standard mouse diet. The tumours in those in the HCCMF group were treated with Huh7 cells when they were five-seven weeks old. The tumors were removed in cases of excessive burden.
The tumors of the three groups showed different growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, the tumours which were treated by RCF didn't show signs of shrinkage. The difference was substantial. The tumors treated with RCF were able to show necrosis, which is common in tumours that are exposed to RCF. phone emf blocker is possible that this necrosis is caused by a lack of oxygen in the more invasive tumors.
In conclusion, the findings show that AM RF EMF is a powerful source of anticancer activity in vitro and in the vivo. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. It is possible that AM RF EMF causes these effects due to CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the growth of HCC tumours in vivo.