EMF Blocking and even EMF Shielding
No matter if you live in a house or apartment or just would like to ensure your home is free of electromagnetic fields There are a variety of ways you can reduce exposure. One of the easiest is to restrict your use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from reaching your house. Another method to protect your house against EMF radiations is to use a RF shielding canopy. It is a type made of net that has EMF shielding. It's used to stop EMFs from entering a space. Another option is to have your house equipped with a conductive enclosure. They are referred to as Faraday cages.
Numerous studies have demonstrated studies have shown that nonionizing EMF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This could be the reason for the long-term responses seen in patients suffering from advanced HCC. However, the mechanism of AM RF EMF's effect in patients suffering from cancer isn't yet clear.
Aspects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumours were divided into three groups. First, the group that was unaffected to RF EMF. Second group members were exposed RF EMF at a frequency that is similar to that used in humans. Third group members were exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumours was compared to that of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage in the tumour.
Click here for tumor-specific AM RF EMF may be based on the fact that cancer cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is mediated by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H may have broader implications for treatment and diagnosis of many cancers.
emf blockers of the control group were not exposed EMF from RF, and fed a normal diet of mice. The tumors in those in the HCCMF group were treated with Huh7 cells after they were between five and seven weeks old. The tumors were then killed in cases of excessive burden.
emf blocking from the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, tumors that were treated using RCF did not show any reduction in size. The difference was significant. The tumors treated with RCF showed necrosis, which is typical in tumors that have been that are exposed to RCF. There is a possibility that this necrosis was due to a lack of oxygen in the more invasive tumours.
In summary, the results indicate that AM RF EMF has anticancer activity in vitro and in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. There is a possibility that AM RF EMF triggers these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the development of HCC tumours in living tissue.