EMF Blocking and EMF Shielding
Whether you live in an apartment or a house, or you just would like to ensure your home is free of electromagnetic fields There are plenty of methods to reduce exposure. One of the easiest is to reduce your use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from reaching your home. block emf to protect your house against EMF radiations is to install a RF shielding canopy. It is a type of net that contains EMF shielding and is used to block EMFs from entering a space. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation in cancer-related stem cells. This could explain the long-term response seen in certain patients suffering from advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not yet clear.
https://anotepad.com/notes/6f29k4is of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumours were divided in three different groups. The first group was not exposed to RF EMF. Second group members were exposed RF EMF at frequencies similar to the one used by humans. In the third, they were subjected the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results revealed that tumours treated with HCCMF showed significant shrinkage. However, tumours treated with RCF did not show any evidence of tumour shrinkage.
The mechanism of tumor-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. https://heronkidney4.bloggersdelight.dk/2023/04/12/emf-blocking-and-emf-shielding/ upon HCC cells is controlled through CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H may have broader implications in the treatment and diagnosis of many cancers.
The tumours in the controls were never exposed EMF from radiofrequency, and fed a standard mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumours from the three groups also displayed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, the tumours which were treated by RCF didn't show signs of shrinkage. The difference was significant. The tumors treated with RCF had necrosis, which is typical in tumors that have been that are exposed to RCF. The possibility is that the necrosis was due to a lack of oxygen in the larger tumors.
In sum, the results suggest the fact that AM EMF has anticancer activity in vitro and in live. Several studies have shown the fact that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the development of HCC tumors in the vivo.