EMF Blocking and EMF Shielding
No matter if you live in a house or apartment or simply want to keep your home free of EMFs There are plenty of ways you can reduce exposure. One of the most effective is to reduce your use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from entering your house. Another way to shield your home against EMF radiation is to put up an RF shielding canopy. This is a fabric made of net which contains EMF shielding. It is utilized to prevent EMFs from entering a space. emf blockers is to get your home fitted with a conductive enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF produces antiproliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could explain the long-term effects observed in patients suffering from advanced HCC. However, the mechanism of AM RF EMF's effect in patients with cancer is not yet clear.
Effects of AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. The first group was not exposed to RF EMF. The second group was exposed to RF EMF at the same frequency to the one used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that tumors treated by HCCMF showed significant shrinkage. However, the tumors treated with RCF did not show any evidence of tumour shrinkage.
The reason for tumour-specific AM RF EMF could be due to the fact that tumour cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused through CACNA1H the protein which mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of many cancers.
The tumours of the controls were never exposed RF EMF, and were fed a normal mouse diet. emf blocker in the HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumours from the three groups showed different growth curves. The HCCMF-treated tumors saw a significant reduction in size of the tumor after eight weeks. However, tumors which were treated by RCF didn't show reduction in size. The difference was significant. emf blocker treated with RCF showed necrosis, which is typical in tumours that are exposed to RCF. The possibility is that the necrosis is caused by a lack of oxygen in the larger tumours.
In conclusion, the findings suggest the fact that AM EMF has anticancer activity in vitro and in vivo. Several studies have shown that AM RF EMF produces measurable reduction in tumours in HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in the vivo.