EMF Blocking and EMF Shielding
If you reside in a house or apartment, or you just would like to ensure your home is free of electromagnetic fields There are plenty options to limit your exposure. One of the easiest is to reduce your electronic device use. emf blockers can also turn to EMF block paint to prevent EMF radiation from entering your house. Another easy way to protect your home against EMF radiation would be to put up a RF shielding canopy. This is a cloth made of net that has EMF shielding and is used to block EMFs from entering a room. Another alternative is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Several studies have shown studies have shown that nonionizing EMF can cause antiproliferative effects on HCC cells. emf blocking of AM RF EMF's anticancer activity in vitro is believed to result from the deregulation the cancer stem cell. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, the mechanism behind AM EMF's effects on patients with cancer is not clear.
The effects from AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were split into three groups. First, block emf that was unaffected to RF EMF. The second group was exposed RF EMF at the same frequency to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results revealed that tumors treated by HCCMF had significant shrinkage. However, tumours treated with RCF didn't show evidence of tumour shrinkage.
The mechanism of tumor-specific AM RF EMF might be due to the fact that tumor cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is controlled by CACNA1H, a protein which mediates tumour-specific Ca2+ influx. The results suggest that CACNA1H could have wider implications in the treatment and diagnosis of many cancers.
The tumors in the control group were not exposed RF EMF, and were fed a standard mouse diet. The tumors of those in the HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were removed when they showed excessive burden.
The tumours in the three groups showed different growth curves. The tumors treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, tumors treated with RCF did not show any shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis, which is common when tumors are that are exposed to RCF. There is a possibility that the necrosis was due to a lack of oxygen in larger tumours.
In sum, the results indicate an AM-RF EMF is a powerful source of anticancer activity in vitro and in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H which is a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the development of HCC tumors in the vivo.