EMF Blocking and EMF Shielding
If you reside in an apartment or house or just want to keep your home free from EMFs There are plenty of methods to reduce exposure. One of the easiest is to reduce your use of electronic devices. You can also turn to EMF blocker paint to block EMF radiation from reaching your house. Another method to protect your house against EMF radiation is to use a shielding canopy for RF. It is a type of net that contains EMF shielding. It's used to stop EMFs from entering a space. block emf is to get your home equipped with an enclosure that is conductive. These devices are called Faraday cages.
Numerous studies have demonstrated how the non-ionizing energy of RF can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to involve down-regulation in cancer-related stem cells. This may account for the long-term effects observed in patients suffering from advanced HCC. However, block emf behind AM EMF's effects on patients with cancer is not yet clear.
The effects on the effects of AM RF EMF on HCC tumour growth in vivo was studied in mice. The tumours were divided in three different groups. First, the group that was unaffected RF EMF. Second group members were exposed RF EMF at frequencies similar to that used in humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results showed that the cancers treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The reason for tumor-specific AM RF EMF could be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused through CACNA1H, a protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H may have broader implications for treatment and diagnosis of many cancers.
The tumours in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumours in HCCMF HCCMF group were treated with Huh7 cells after they were five-seven weeks old. The tumors were removed when they showed excessive burden.
The tumours from the three groups showed distinct growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumors treated with RCF did not show any reduction in size. The difference was highly significant. The tumors treated by RCF had necrosis, which is common when tumors are that are exposed to RCF. The possibility is that the necrosis is caused by the lack of oxygen in the larger tumors.
In conclusion, the findings indicate an AM-RF EMF is a powerful source of anticancer activity in vitro and in vivo. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, block emf may have a long-lasting effect on the development of HCC tumours in vivo.