EMF Block Paint plus EMF Shielding
Whether you live in an apartment or house or simply would like to keep your home free of EMFs, there are a number of ways you can reduce exposure. One of the easiest is to limit the use of electronic devices. You could also consider EMF block paint to stop EMF radiation from entering your house. Another method to protect your home from EMF radiation is to put up an RF shielding cover. This is a cloth of net that has EMF shielding. It is utilized to stop EMFs from entering a space. Another alternative is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
Several studies have shown studies have shown that the nonionizing EMF can cause antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could be the reason for the long-term effects observed in certain patients suffering from advanced HCC. However, emf blocking behind AM RF EMF's effect in cancer patients is not evident.
The effects on the effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. https://canvas.instructure.com/eportfolios/2010950/Home/Details_of_Emf_Blocking_Radiation_2 were classified in three different groups. One group did not have exposure RF EMF. Second group members were exposed RF EMF at the same frequency to that of humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumors was evaluated against that of RCF. The results indicated that tumors treated by HCCMF showed significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. Homepage indicate that CACNA1H may have broader implications for the diagnosis and treatment of a variety of cancers.
The tumors in the controls were never exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumors in the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed different growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumours treated with RCF did not show any signs of shrinkage. The difference was significant. The tumors treated with RCF showed necrosis, which is typical in tumours that are exposed to RCF. The possibility is that the necrosis is caused by an absence of oxygen in the larger tumors.
In sum, the results show an AM-RF EMF has anticancer effects in vitro and in vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF triggers these effects through CACNA1H which is a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in living tissue.