EMF Block Paint plus EMF Shielding
If you reside in an apartment or a house or simply want to keep your home free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most straightforward is to restrict your electronic device use. It is also possible to use EMF blocker paint to block EMF radiation from reaching your home. Another way to shield your house from EMF radiation is to put up an RF shielding canopy. It's a kind of net that contains EMF shielding. It's used to block EMFs from entering a room. Another alternative is to have your home equipped with an electrical enclosure. These enclosures are known as Faraday cages.
emf blockers of studies have proven studies have shown that the nonionizing EMF produces anti-proliferative effects on HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. emf blocking may account for the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in patients with cancer is not evident.
Aspects from AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were classified into 3 groups. The first group was not exposed to RF EMF. Another group of participants was subjected RF EMF at the same frequency to that of humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results revealed that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect on HCC cells is caused through CACNA1H, a protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H may have broader implications in the diagnosis and treatment of various cancers.
The tumours in the control group were not exposed to EMF from RF, and fed a normal mouse diet. The tumours in the HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumors were removed in cases of excessive burden.
The tumors of the three groups also showed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in tumour size after 8 weeks. However, tumors that were treated using RCF did not show any reduction in size. The difference was highly significant. The tumors treated with RCF were able to show necrosis, which is common in tumors that have been exposed to RCF. It is possible that the necrosis was caused by a lack of oxygen in the more invasive cancers.
In sum, the results suggest that AM RF EMF exhibits anticancer activity in vitro and in live. A number of studies have proven that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF produces these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting influence on the development of HCC tumors in living tissue.