EMF Block Paint in addition to EMF Shielding
No matter if you live in an apartment or house, or you just want to keep your home free from EMFs There are a variety of methods to reduce exposure. One of the most straightforward is to limit the use of electronic devices. https://k12.instructure.com/eportfolios/367651/Home/EMF_Block_Paint_and_even_EMF_Shielding could also consider EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiations is to use an RF shielding cover. This is a cloth made of net which contains EMF shielding and is used to block EMFs from entering rooms. Another alternative is to have your house equipped with a conductive enclosure. They are referred to as Faraday cages.
Numerous studies have demonstrated how the EMF that is not ionized has anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This could be the reason for the long-term effects observed in certain patients suffering from advanced HCC. But, emf prevention for AM EMF's impact on patients with cancer is not clear.
Effects from AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumors were classified in three different groups. The first group was not exposed to RF EMF. Another group of participants was subjected to RF EMF at a frequency that is similar to that used in humans. Third group members were exposed the RF EMF in HCC-specific frequencies. The effect of HCCMF on the tumours was compared to that of RCF. The results indicated that tumors treated by HCCMF had significant shrinkage. However, the tumours treated with RCF didn't show evidence of shrinkage in the tumour.
The mechanism of tumour-specific AM RF EMF may be due to the fact that cancer cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is mediated by CACNA1H, a protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have wider implications in the diagnosis and treatment of a variety of cancers.
The tumours of the control group were not exposed to EMF from radiofrequency, and fed a normal mouse diet. The tumors in the HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups showed distinct growth curves. The HCCMF-treated tumors had a significant reduction in the size of the tumour after eight weeks. However, the tumours that were treated using RCF showed no reduction in size. The difference was significant. The tumors treated by RCF had necrosis, which is typical in tumors that have been exposed to RCF. Homepage is possible that this necrosis was due to an absence of oxygen in larger tumors.
In summary, the results show that AM RF EMF exhibits anticancer activity in vitro and in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may cause a lasting effect on the growth of HCC tumors in the vivo.