EMF Block Paint in addition to EMF Shielding
Whether you live in an apartment or a house, or you just would like to keep your home free of EMFs There are a variety of methods to reduce exposure. One of the easiest is to reduce the usage of your electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your house. Another way to shield your home against EMF radiations is to put up a RF shielding canopy. It's a kind made of net which contains EMF shielding and is used to block EMFs from entering a space. Another option is to get your home fitted with a conductive enclosure. These enclosures are known as Faraday cages.
Several studies have shown that the non-ionizing RF EMF produces antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation in cancer-related stem cells. This could explain the long-term response seen in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in cancer patients isn't clear.
Aspects from AM RF EMFs on HCC tumor growth in vivo was studied in mice. Discover more were divided into three groups. One group did not have exposure to RF EMF. emf blocker were exposed to RF EMF at a frequency that is similar to that of humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results showed that the tumours treated with HCCMF were significantly shrinking. However, the tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism of tumour-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects on HCC cells is mediated by CACNA1H the protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H may have broader implications for diagnosis and treatment of a variety of cancers.
The tumours in the controls were never exposed EMF from RF, and fed a normal mouse diet. The tumors of the HCCMF group were injected with Huh7 cells after they were 5 to 7 weeks old. The tumours were then euthanized after they had a high burden.
The tumors in the three groups showed different growth curves. The tumours treated with HCCMF showed a significant decrease in tumour size after 8 weeks. However, tumors that were treated using RCF didn't show shrinkage. The difference was significant. emf blocking treated by RCF showed necrosis, which is common in tumors that have been exposed to RCF. It is possible that the necrosis was caused by the lack of oxygen in the larger cancers.
In conclusion, the findings indicate an AM-RF EMF is a powerful source of anticancer effects in vitro and in live. A number of studies have proven it is true that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF produces these effects due to CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained effect on the development of HCC tumors in the vivo.