EMF Block Paint in addition to EMF Shielding
No matter if you live in an apartment or a house or just would like to keep your home free from EMFs, there are a number options to limit your exposure. One of the easiest is to reduce the use of electronic devices. You could also consider EMF blocker paint to block EMF radiation from entering your home. Another way to shield your house from EMF radiation would be to install an RF shielding canopy. It is a type of net that has EMF shielding. It's used to prevent EMFs from entering a space. Another option is to get your house equipped with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown how the EMF that is not ionized has antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed involve down-regulation in cancer-related stem cells. emf blockers may account for the long-term effects observed in certain patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in patients with cancer is not clear.
Aspects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo was studied in mice. The tumors were classified into three groups. First, the group that was unaffected RF EMF. https://hastings-mouritsen.mdwrite.net/emf-blocking-plus-emf-shielding-1680927296 were exposed to RF EMF at a frequency that is similar to that used in humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumors was assessed against the effect of RCF. The results showed that the cancers treated with HCCMF had significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF might be driven by the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. https://anotepad.com/notes/b74dq3hg on HCC cells is mediated through CACNA1H, a protein which regulates the Ca2+ influx specific to tumors. The findings suggest that CACNA1H could have more broader implications for treatment and diagnosis of various cancers.
The tumors in those in the group that were unaffected EMF from RF, and fed a standard mouse diet. The tumours in the HCCMF group were infected with Huh7 cells at the time they were between five and seven weeks old. The tumors were removed after they had a high burden.
The tumors of the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in tumour size after eight weeks. However, the tumors that were treated using RCF showed no shrinkage. The difference was significant. The tumors treated with RCF were able to show necrosis, which is typical in tumors that have been exposed to RCF. It is possible that this necrosis is caused by a lack of oxygen in larger tumours.
In sum, the results suggest an AM-RF EMF exhibits anti-cancer properties in vitro as well as in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable tumour shrinkage within HCC patients. The possibility is that the AM EMF produces these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the growth of HCC tumours in vivo.