EMF Block Paint in addition to EMF Shielding
If you reside in an apartment or a house or just need to ensure that your house is free of EMFs, there are a number of methods to reduce exposure. One of the most straightforward is to reduce the usage of your electronic devices. You can also turn to EMF block paint to stop EMF radiation from reaching your home. Another easy way to protect your house from EMF radiation would be to put up a RF shielding canopy. It is a type of net that contains EMF shielding. It is utilized to prevent EMFs from entering a room. Another alternative is to have your home fitted with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven studies have shown that the nonionizing EMF produces antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This may account for the long-term response seen in patients with advanced HCC. However, the reason behind AM RF EMF's effect in cancer patients is not evident.
The effects on the effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumors were divided into three groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at frequencies similar to that used in humans. Third group members were exposed RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumors was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF didn't show evidence of tumour shrinkage.
The mechanism of tumour-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is mediated through CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications for the diagnosis and treatment of a variety of cancers.
The tumours in those in the group that were unaffected EMF from radiofrequency, and fed a standard mouse diet. The tumors in the HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumours were then euthanized when they showed excessive burden.
The tumours in the three groups also showed distinct growth curves. The tumours treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, the tumors which were treated by RCF showed no shrinkage. The difference was significant. emf blockers treated with RCF had necrosis, which is typical in tumours that are exposed to RCF. There is block emf that this necrosis is caused by the lack of oxygen in the larger tumors.
In summary, the results show that AM RF EMF exhibits anti-cancer properties in vitro as well as in the vivo. emf blocking of studies have proven that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein that is involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the growth of HCC tumors in vivo.