EMF Block Paint and EMF Shielding
No matter if you live in an apartment or a house, or you just need to ensure that your house is free of electromagnetic fields There are a variety of methods to reduce exposure. One of the most effective is to reduce the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your house. Another easy way to protect your house against EMF radiations is to install an RF shielding cover. It is a type of net that has EMF shielding. It is utilized to stop EMFs from entering a room. Another option is to have your home fitted with an electrical enclosure. emf blocker are called Faraday cages.
Several studies have shown how the EMF that is not ionized produces antiproliferative effects in HCC cells. https://ctxt.io/2/AACQ7U05Ew behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This could explain the long-term response seen in certain patients suffering from advanced HCC. However, the reason behind AM EMF's impact on patients with cancer is not yet clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo was studied in mice. The tumours were separated into 3 groups. The first group was not exposed to RF EMF. https://anotepad.com/notes/nm745668 were exposed RF EMF at the same frequency to that of humans. The third group was exposed RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumors was compared to that of RCF. The results indicated that cancers treated with HCCMF showed significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is caused through CACNA1H the protein which mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have more broader implications for the diagnosis and treatment of various cancers.
The tumors in the controls were never exposed to RF EMF, and were fed a normal mouse diet. The tumors of HCCMF HCCMF group were treated with Huh7 cells at the time they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumors of the three groups also displayed distinct growth curves. The tumors treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, tumors that were treated using RCF did not show any signs of shrinkage. The difference was highly significant. The tumors treated by RCF had necrosis, which is common when tumors are exposed to RCF. It is possible that this necrosis was due to an absence of oxygen in the larger tumors.
In summary, the results indicate an AM-RF EMF exhibits anti-cancer properties in vitro as well as in live. Several studies have shown the fact that AM RF EMF produces measurable shrinkage of tumors for HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting effect on the growth of HCC tumors in the vivo.