EMF Block Paint and EMF Shielding
If you reside in an apartment or a house or simply want to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to limit the usage of your electronic devices. emf blocker could also consider EMF block paint to prevent EMF radiation from reaching your house. Another way to shield your house from EMF radiations is to use an RF shielding canopy. This is a cloth of net that contains EMF shielding and is used to prevent EMFs from entering a space. Another option is to have your house equipped with an electrical enclosure. emf blocking are referred to as Faraday cages.
Several studies have shown how the EMF that is not ionized can cause antiproliferative effects in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to result from the deregulation in cancer-related stem cells. This may account for the long-term response seen in some patients with advanced HCC. However, the reason behind AM RF EMF's effect in patients with cancer is not clear.
The effects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumours were divided in three different groups. One group did not have exposure to RF EMF. Second group members were exposed to RF EMF at frequencies similar to that used in humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on the tumours was evaluated against that of RCF. The results revealed that tumors treated by HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of shrinkage in the tumour.
emf blocking behind tumor-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 type voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is mediated by CACNA1H, a protein that is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have more broader implications in the diagnosis and treatment of a variety of cancers.
The tumours in the controls were never exposed EMF from radiofrequency, and fed a standard mouse diet. The tumours in HCCMF HCCMF group were infected with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed when they showed excessive burden.
The tumours in the three groups showed different growth curves. The HCCMF-treated tumors had a significant reduction in the size of the tumour after eight weeks. However, tumors that were treated using RCF did not show any shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis that is common in tumours exposed to RCF. The possibility is that this necrosis is caused by the lack of oxygen in the larger cancers.
In summary, the results indicate the fact that AM EMF exhibits anti-cancer properties in vitro as well as in live. A number of studies have proven it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. There is a possibility that AM RF EMF causes these effects because of CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in the vivo.