EMF Block Paint and EMF Shielding
No matter if you live in an apartment or house, or you just would like to keep your home free from EMFs, there are a number options to limit your exposure. One of the easiest is to reduce the usage of your electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your house. Another way to shield your home against EMF radiation is to install an RF shielding canopy. This is a cloth of net that has EMF shielding. It is utilized to prevent EMFs from entering a room. Another alternative is to have your house equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.
Numerous studies have demonstrated how the EMF that is not ionized can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed be based on the downregulation in cancer-related stem cells. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, the reason behind AM EMF's impact on patients with cancer is not clear.
Aspects on the effects of AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumours were classified into three groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at a frequency that is similar to the frequency used by humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumors treated by HCCMF showed significant shrinkage. However, tumours treated with RCF showed no evidence of shrinkage in the tumour.
The mechanism behind tumour-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 voltage calcium channels for proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is mediated by CACNA1H the protein that mediates tumour-specific Ca2+ influx. The results indicate that CACNA1H could have more broader implications for treatment and diagnosis of various cancers.
The tumours in the controls were never exposed EMF from radiofrequency, and fed a standard mouse diet. The tumours in the HCCMF group were treated with Huh7 cells after they were 5 to 7 weeks old. emf blocking were then killed when they showed excessive burden.
The tumors in the three groups also displayed distinct growth curves. The tumors treated with HCCMF saw a significant decrease in size of the tumor after 8 weeks. However, the tumours that were treated using RCF showed no reduction in size. The difference was significant. emf blocker treated with RCF showed necrosis, which is common in tumors that have been exposed to RCF. There is a possibility that this necrosis was caused by an absence of oxygen in larger tumors.
In summary, the results show an AM-RF EMF is a powerful source of anticancer activity in vitro and in the vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF produces these effects through CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the development of HCC tumours in the vivo.