EMF Block Paint and EMF Shielding
No matter if you live in an apartment or house or just want to keep your home free of electromagnetic fields There are plenty of ways you can reduce exposure. One of the easiest is to reduce the usage of your electronic devices. You can also turn to EMF block paint to prevent EMF radiation from reaching your home. Another way to shield your house against EMF radiation would be to use an RF shielding cover. It's a kind of net that contains EMF shielding. It's used to stop EMFs from entering a room. Another option is to have your home equipped with an electrical enclosure. These enclosures are known as Faraday cages.
Several studies have shown how the EMF that is not ionized produces antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This could be the reason for the long-term response seen in patients with advanced HCC. But, the reason for AM EMF's effects on patients with cancer is not clear.
The effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. http://note.pearltrees.com/13949600/344801174f55409ab98c6c704cc67701 were divided into three groups. First, the group that was unaffected to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. The third group was exposed RF EMF with HCC-specific modulation frequencies. emf blockers of HCCMF on the tumours was evaluated against that of RCF. The results indicated that tumors treated by HCCMF were significantly shrinking. However, the tumors treated with RCF didn't show evidence of shrinkage of the tumor.
The mechanism behind tumor-specific AM RF EMF might be due to the fact that cancer cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation in HCC cells is caused by CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications in the diagnosis and treatment of a variety of cancers.
The tumours of the control group were not exposed to EMF from RF, and fed a standard mouse diet. The tumors of HCCMF HCCMF group were infected with Huh7 cells after they were between five and seven weeks old. The tumors were then killed when they showed excessive burden.
emf blocker in the three groups also displayed distinct growth curves. The HCCMF-treated tumors had a significant reduction in tumour size after eight weeks. However, the tumours which were treated by RCF showed no signs of shrinkage. The difference was significant. The tumours treated with RCF had necrosis, which is typical in tumours exposed to RCF. There is a possibility that this necrosis was due to an absence of oxygen in larger tumours.
In conclusion, the findings suggest an AM-RF EMF has anti-cancer properties in vitro as well as in the vivo. Numerous studies have demonstrated it is true that AM RF EMF produces measurable reduction in tumours within HCC patients. It is possible that AM RF EMF causes these effects through CACNA1H, a protein involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting impact on the development of HCC tumours in vivo.