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Путешественницы Черногория

Please note that there are more than genetic variants of the deficiency. G6PD deficiency is most common in males of African heritage. Many females of African heritage are carriers of G6PD deficiency, meaning they can pass the gene for the deficiency to their children but do not have symptoms; only a few are actually affected by G6PD deficiency. People of Mediterranean heritage, including those of Italian, Greek, Arabic, and Sephardic Jewish backgrounds, also are commonly affected. The severity of G6PD deficiency varies among these groups — it tends to be milder in those of African heritage and more severe in people of Mediterranean descent. Why does G6PD deficiency happen more often in certain groups of people? It is known that Africa and the Mediterranean basin are high-risk areas for the infectious disease malaria. Researchers have found evidence that the parasite that causes this disease does not survive well in G6PD-deficient cells. So they believe that the deficiency may have developed as a protection against malaria. The frequency quoted in Pakistan is between This study shows the frequency to be 4. O data Mehmood, A. When the condition is identified through newborn screening and properly managed, children with G6PD deficiency often can lead healthy lives. Rarely, kidney failure or death may occur following a severe hemolytic event. G6PD is one of many enzymes that help the body process carbohydrates and turn them into energy. G6PD also protects red blood cells from potentially harmful byproducts that can accumulate when a person takes certain medications or when the body is fighting an infection. Hemolytic anemia occurs when the bone marrow the soft, spongy part of the bone that produces new blood cells cannot compensate for this destruction by increasing its production of RBCs. This deficiency can cause hemolytic anemia, usually after exposure to certain medications, foods, or even infections. In these cases, the symptoms disappear once the cause, or trigger, is removed. In rare cases, G6PD deficiency leads to chronic anemia. G6PD deficiency is passed along in genes from one or both parents to a child. The gene responsible for this deficiency is on the X chromosome. A child with G6PD deficiency who is exposed to a medication or infection that triggers the destruction of RBCs may have no symptoms at all. In more serious cases, a child may exhibit symptoms of hemolytic anemia also known as a hemolytic crisis , including:. Once the trigger is removed or resolved, the symptoms of G6PD deficiency usually disappear fairly quickly, typically within a few weeks. The best way to care for a child with G6PD deficiency is to limit exposure to the triggers of its symptoms. With the proper precautions, G6PD deficiency should not keep your child from living a healthy, active life. If symptoms are mild, no medical treatment is usually needed. As the body naturally makes new red blood cells, the anemia will improve. If symptoms are more severe, a child may need to be hospitalized for supportive medical care. The main treatment for G6PD deficiency is avoidance of oxidative stressors. Rarely, anemia may be severe enough to warrant a blood transfusion. Splenectomy generally is not recommended. Folic acid and iron potentially are useful in hemolysis, although G6PD deficiency usually is asymptomatic and the associated hemolysis usually is short-lived. Antioxidants such as vitamin E and selenium have no proven benefit for the treatment of G6PD deficiency. Research is being done to identify medications that may inhibit oxidative-induced hemolysis of G6PD-deficient red blood cells. The best therapy is simply to avoid the prohibited drugs and foodstuffs. In case of a hemolytic crisis, the most effective therapy is blood transfusion. In milder cases of the deficiency, transfusion is usually not required. Any boy suffering from it should be informed of his deficiency so he can help himself avoid the intake of the forbidden foodstuff in various situations where he is not under your supervision such as school. You should also inform school supervisors and, where applicable, also the school caterers who provide school lunch. March 31, March 31, By Danielle Wilson. It has been suggested that G6PD deficiency offers some protection against malaria. Local The frequency quoted in Pakistan is between What is G6PD? Outlook When the condition is identified through newborn screening and properly managed, children with G6PD deficiency often can lead healthy lives. Symptoms A child with G6PD deficiency who is exposed to a medication or infection that triggers the destruction of RBCs may have no symptoms at all. In more serious cases, a child may exhibit symptoms of hemolytic anemia also known as a hemolytic crisis , including: paleness in darker-skinned kids, paleness is sometimes best seen in the mouth, especially on the lips or tongue extreme tiredness rapid heartbeat rapid breathing or shortness of breath jaundice, or yellowing of the skin and eyes, particularly in newborns an enlarged spleen dark, tea-colored urine Sudden rise of body temperature Once the trigger is removed or resolved, the symptoms of G6PD deficiency usually disappear fairly quickly, typically within a few weeks. Avoid the intake of any drugs. You would most probably be requested to list all foodstuffs and drugs taken in the preceding 48 hours, so try to recall and list them. C 3 Neonates should be screened for G6PD deficiency when family history, ethnic or geographic origin, or the timing of the appearance of neonatal jaundice suggests the possibility of G6PD deficiency. C 23 G6PD can be diagnosed with a quantitative spectrophotometric analysis or, more commonly, by a rapid fluorescent spot test. Confirmation Other tests that may be done include: Bilirubin level Complete blood count, including red blood cell count Hemoglobin — blood Hemoglobin — urine Haptoglobin level LDH test Methemoglobin reduction test Reticulocyte count Treatment The best way to care for a child with G6PD deficiency is to limit exposure to the triggers of its symptoms. Obtain your vitamins and minerals from natural sources such as bone stock soups. Round out your diet with proteins and carbohydrates not found on the G6PD Deficiency forbidden foods list. Do not consume foods or supplements that contain large amounts of VItamin C, also called ascorbic acid, or artificial blue dyes. Do not eat menthol, or menthol-containing foods such as breath mints or candy. Do not eat legumes, such as lima beans, fava beans, kidney beans or soybeans. Avoid any soy-containing products, such as tofu, bean curds, miso or textured soy protein. Look out for less easily recognizable legumes such as alfalfa sprouts, peanuts, licorice or carob. Some legumes — such as fava beans — cause severe hemolysis — but several other legumes, beans and pulses cause less severe hemolysis that, while mild enough to evade initial detection, can still damage your health. Avoidance: Avoid already-prepared Chinese foods. These foods can contain herbs that could provoke a hemolytic episode. These herbs include Flos Chimonanthi praecosis, Rhizoma coptidis, margarita, Flos ionicerae and Calculus bovis. Furthermore, avoid packaged foods that might contain forbidden food ingredients. These foods include margarine, jarred salad dressings, canned soups, potato chips, canned fish and meats, hot dogs, sausages, processed meats, lowfat cheeses and sauces such as sweet and sour sauce or Worcestershire sauce. Warning: Ask your doctor for an updated G6PD deficiency forbidden foods list every six months to ensure that you do not continue to eat foods shown to initiate hemolytic episodes. Your doctor might also suggest that you hire a registered dietitian familiar with this disorder to better ensure that your nutritional needs are met in relation to your other current medical conditions. Therapies The best therapy is simply to avoid the prohibited drugs and foodstuffs. Advice for Parents Any boy suffering from it should be informed of his deficiency so he can help himself avoid the intake of the forbidden foodstuff in various situations where he is not under your supervision such as school. Leave a Reply Cancel Reply Your email address will not be published. Patients with G6PD deficiency should avoid exposure to oxidative drugs Table 3 and ingestion of fava beans. Neonates should be screened for G6PD deficiency when family history, ethnic or geographic origin, or the timing of the appearance of neonatal jaundice suggests the possibility of G6PD deficiency. G6PD can be diagnosed with a quantitative spectrophotometric analysis or, more commonly, by a rapid fluorescent spot test. Antibiotic used in combination preparations i.