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Introduction: The coronavirus disease COVID pandemic provoked unprecedented disturbance in hypertension care, while alarming concerns arose about its long-term consequences. We investigated the trends of emergency visits and admissions regarding uncontrolled hypertension in order to assess the impact of COVID spread on population behavior towards hypertension urgencies during its first wave. Material and methods: Data from daily unscheduled visits and admission counts in the Cardiology sector were collected from the Emergency Department database of a tertiary General Hospital in Athens, Greece for the period January 15th to July 15th These data were compared with those from the previous year. Cases of patients who presented with hypertensive urgency or who were admitted due to uncontrolled hypertension were separately analyzed. Results: A total of 7, patient records were analyzed. This is a complex phenomenon, closely related to increased levels of public stress, disruptions in health care services and to a lesser extent to the imposed restrictions in transportation. The initial relative increase in uncontrolled hypertension-related admissions rate, combined with the later increase of hypertensive urgencies may be indicative of blood pressure deregulation among the studied population, which is multifactorial and potentially detrimental. To better understand the global pattern of this relationship, we conducted a meta-analysis on endothelial biomarkers related to COVID severity. Pooled estimates and mean differences PMD for each biomarker were reported. These findings strengthen the evidence on the key role of endothelial dysfunction in disease progress. The hypothesis was that angiotensin-converting enzyme inhibitor ACEIs or angiotensin receptor blocker ARBs might up-regulate ACE2 expression that is used as a receptor for viral entry into cells. Several observational studies arrive at the conclusion that the continued use of RAS inhibitors is unlikely to be harmful in COVIDpositive patients. Importance: Severe acute respiratory syndrome coronavirus 2 infection has evolved into a global pandemic. Low-dose colchicine combines anti-inflammatory action with a favorable safety profile. Objective: To evaluate the effect of treatment with colchicine on cardiac and inflammatory biomarkers and clinical outcomes in patients hospitalized with coronavirus disease COVID Design, Setting, and Participants: In this prospective, open-label, randomized clinical trial the Greek Study in the Effects of Colchicine in COVID Complications Prevention , patients hospitalized with COVID were randomized in a allocation from April 3 to April 27, , to either standard medical treatment or colchicine with standard medical treatment. The study took place in 16 tertiary hospitals in Greece. Intervention: Colchicine administration 1. Main Outcomes and Measures: Primary end points were 1 maximum high-sensitivity cardiac troponin level; 2 time for C-reactive protein to reach more than 3 times the upper reference limit; and 3 time to deterioration by 2 points on a 7-grade clinical status scale, ranging from able to resume normal activities to death. Secondary end points were 1 the percentage of participants requiring mechanical ventilation, 2 all-cause mortality, and 3 number, type, severity, and seriousness of adverse events. The primary efficacy analysis was performed on an intention-to-treat basis. Results: A total of patients were evaluated 61 \\\\\\\\[ Median interquartile range peak high-sensitivity cardiac troponin values were 0. Median interquartile range maximum C-reactive protein levels were 4. The clinical primary end point rate was Mean SD event-free survival time was Adverse events were similar in the 2 groups, except for diarrhea, which was more frequent with colchicine group than the control group 25 patients \\\\\\\\[ Conclusions and Relevance: In this randomized clinical trial, participants who received colchicine had statistically significantly improved time to clinical deterioration. There were no significant differences in high-sensitivity cardiac troponin or C-reactive protein levels. These findings should be interpreted with caution. Trial Registration: ClinicalTrials. Among its potential mechanisms of action is the non-selective inhibition of NLRP3 inflammasome which is thought to be a major pathophysiologic component in the clinical course of patients with COVID PaO2 Основное содержание 1 Поиск 2 нижний колонтитул 3. Главная Поиск au:'Tousoulis, Dimitrios' 12 Отсортировать Дата поступления по убыванию Дата поступления увеличение Актуальность по годам - сначала новые - по годам - сначала старые -. Шоу: 20 50 Результаты 1 - 12 de Добавить фильтры. Основная тема. Показать еще Тип исследования. Годовой диапазон. Hypertensive urgencies during the first wave of the COVID pandemic in a tertiary hospital setting: a U-shaped alarming curve. Arch Med Sci ; 18 4 : , Реферат Introduction: The coronavirus disease COVID pandemic provoked unprecedented disturbance in hypertension care, while alarming concerns arose about its long-term consequences. A Systematic Review and Meta-analysis. Curr Med Chem ; 29 21 : , Curr Hypertens Rev ; 18 1 : , Hellenic J Cardiol ; 62 5 : , Clin Res Cardiol ; 12 : , Dec. Реферат The original version of this article unfortunately contained a mistake. Hellenic J Cardiol ; 62 3 : , Unveiling the mystery of 'missing' emergencies in the era of COVID pandemic: a generalized phenomenon with unclarified implications. Реферат Importance: Severe acute respiratory syndrome coronavirus 2 infection has evolved into a global pandemic. Nicotine Tob Res ; 22 9 : , 08 Hellenic J Cardiol ; 61 1 : , Смотрите больше деталей. Критерии поиска au:'Tousoulis, Dimitrios'. Ваше имя Ваш адрес Отправить. Эту страницу Отмечено 0.
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