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Official websites use. Share sensitive information only on official, secure websites. Advocates for the legalization of medical and retail marijuana are quick to point out all the possible benefits that a community might see from such a venture. The more potent a drug is, the stronger the possibility of addiction and the more likely the person will continue to purchase and use the product. The active component in marijuana that people find so desirable was not really known until the s when a research team in Israel found that after injecting THC into aggressive rhesus monkeys, they became calm and sedate. It was not until the s that this same team discovered why we have these receptors in our brain. The primary problem with the current available cannabis in dispensaries in Colorado is that the THC content is not like it used to be. For example the Girl Scout Cookie strain has only 0. The flower or leaves that are generally smoked or vaped are only one formulation. There is absolutely no research that indicates this level of THC is beneficial for any medical condition. The purpose of these products is to produce a high, and the increased potency makes them potentially more dangerous and more likely to result in addiction. Because there was initially no regulation on the edibles they have been made to look very similar to regular products that people consume such as chocolates, gummy bears, PopTarts etc. Keep away from Children. According to the Monitoring the Future Study, marijuana is by far the number one drug abused by eighth and twelfth graders. However, there are significant consequences of long-term or heavy marijuana use beginning in adolescence. Adolescence is a time of significant brain development. Normally during this period there is a significant increase in dopaminergic and glutamatergic stimulatory neurotransmitters and a decrease in serotonergic and GABAergic suppressive neurotransmitters located in the pre-frontal motor cortex — the last part of the brain to fully develop. This equates to a great deal of learning, exploring and doing during this period, similar to stepping on the gas pedal and problems with impulse control and judgement, similar to problems stepping on the brake. The marijuana of old used to be classified as a hallucinogen and was thought to not cause addiction because there was no identified withdrawal syndrome. This has changed and with the increased potency of THC there is a definite recognized withdrawal syndrome which includes increased anger, irritability, depression, restlessness, headache, loss of appetite, insomnia and severe cravings for marijuana. All drugs of abuse cause a release of dopamine from the nucleus acumbens that signifies salience and starts the process of long term potentiation which reinforces the learning. At the same time, the hippocampus which is vitally important for new memory and learning is negatively impacted by the chronic use of any addictive substance. These substances decrease neurogenesis in the hippocampus and actually cause shrinkage of the hippocampus and impair the ability to learn new things. This is true for alcohol, cocaine, methamphetamine, heroin, nicotine, and THC. When the use of addictive drugs is stopped and the animals are allowed to be in a recovery environment where they are free to exercise voluntary exercise being one thing that improves neurogenesis they can again learn new things. A study of 40 male and 34 female long-term 15 years cannabis users versus 37 non-users, healthy controls divided the marijuana users into three groups; those that smoked predominantly THC in the previous three months, those who smoked a combination of THC and CBD in the previous three months and former uses with a sustained abstinence of 29 months. In the former users the hippocampal integrity was comparable to controls. The only problem with this study is they did not test for functional deficits to see if function improved along with hippocampal volume. There are other important neurotransmitters that are very active during adolescence and include acetylcholine receptors ACH and endocannabinergic receptors CB1. ACH helps us focus and concentrate and ACH innervation of the pre-frontal motor cortex reaches mature levels during adolescence. They are called nicotinic simply because nicotine binds to these receptors — not because we are supposed to use tobacco products. These receptors are involved in promoting or preventing neuronal cell death depending on the stage of brain development. Putting an exogenous form of nicotine in the developing brain, as in consuming tobacco, can dysregulate these fine tuning mechanisms during adolescence. CB1 receptors regulate the balance between excitatory and inhibitory neuronal activity utilizing our own natural anandamides. Exposure to cannabis during adolescence disrupts glutamate which plays an important role in synaptic pruning in the pre-frontal motor cortex; disrupting normal brain development. A study in New Zealand with a year follow-up showed an average loss of 8 IQ points with early persistent teen use of marijuana. A study out of Yale University tracked 1, students who achieved similar SAT scores and were enrolled in college. Those who drank alcohol without using marijuana had an average GPA of 3. Marijuana use is also correlated with creating or worsening many mental health problems including anxiety, depression, psychosis, and suicidal ideation. A prospective study in Australia followed 1, girls for seven years starting before they expressed symptoms of mental illness or substance abuse. A study of adults with depression assessed symptoms, functioning and marijuana use at baseline, and three- and six-month intervals. Numerous studies have demonstrated that using cannabis prior to the age of 15—18 significantly increases the risk of developing psychotic symptoms. A landmark study out of the UK analyzed adults, ages 18—65, with their first psychotic episode versus matched healthy controls. A growing number of states have identified PTSD as an approved condition for medical marijuana. However, this is not based on any research. There is no evidence that marijuana successfully treats PTSD and there is evidence that it can make it worse. All these compounds do is provide temporary relief by numbing the individual and disconnecting them from the traumatic emotion. It does not resolve the trauma, and they have to continue to use multiple times a day in order to continue with the benefit. This can lead to increased addiction potential and withdrawal symptoms, cognitive impairment, a-motivational syndrome, and the potential for psychosis or worsening psychosis from the PTSD. Those who were using marijuana but stopped using it in treatment had the lowest level of PTSD symptoms four months after treatment, and those who started smoking marijuana had the highest levels of violent behavior and PTSD symptoms four months after treatment. Another conundrum that impacts treatment for PTSD is the possibility that cannabis users have an increased susceptibility to memory distortions even when abstinent and drug free which can compromise reality monitoring. Riba et al. The study involved a memory paradigm including a study phase and a testing phase with the participant in an MRI scanner. They were given lists of four words to memorize and then shown a different list and they had to report if the words were on the previous list. Marijuana users were significantly more likely to have false recognition of the words and were less likely to reject that they had a false memory compared with the non-users. Multiple studies have documented a relationship between cannabis use and suicidality. A large, longitudinal study in Australia and New Zealand of over adolescents and maximum frequency of marijuana use found almost a seven fold increase in suicide attempts in daily marijuana users compared with non-users. According to the Colorado Department of Public Health and Environment, marijuana is by far the most frequently encountered drug on toxicology screens of suicides among adolescents ages 10 — 19 and has been increasing over the last eight years. Misguided marijuana advocates have recently been suggesting that marijuana is a solution for the opioid epidemic. A study of 5, adolescents in the UK with three or more measures of cannabis use from age 13—18 found a dose-response relationship between cannabis use trajectories in adolescence and nicotine dependence, harmful alcohol consumption, and other illicit drug use by age There is evidence that prenatal exposure of cannabis can alter opioid gene function in humans. Fetal brains obtained from aborted fetuses from women who were using marijuana during their pregnancy were compared to those from women not using marijuana during pregnancy. These findings are comparable to findings with animals. One study of prenatal cannabis exposure in rats found that the THC exposed rats exhibited shorter latency to first active lever press for heroin and had higher heroin-seeking during mild stress and drug extinction than animals not exposed to THC. Another interesting study that supports the idea that cannabis use and opioid use are linked was in a randomized, double-blind, placebo controlled trial of naltrexone in non-treatment seeking cannabis smokers. If states continue to commercialize marijuana as has been done in Colorado we are destined to see many more people requiring treatment for addiction, depression, anxiety, suicidal ideation, and psychosis. We need to continually educate every one of the risks and increase prevention efforts to prevent children and adolescents from initiating marijuana use. This should include a strong ban on any advertising that appears to be directed toward youth — for all drugs including marijuana, tobacco, and alcohol. States will need to commit to increased funding for and availability of treatment options. The strongest recommendation would be to initiate regulations to limit the concentration of THC. She is the medical director for a inpatient dual diagnosis treatment program in Pueblo, Colorado. As a library, NLM provides access to scientific literature. Mo Med. Find articles by Elizabeth Stuyt. Copyright by the Missouri State Medical Association. Contact: libbystuyt msn. Similar articles. Add to Collections. Create a new collection. Add to an existing collection. Choose a collection Unable to load your collection due to an error Please try again. Add Cancel.

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