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Joost H. Steenwinkel, Jeff G. Occupational exposure to polycyclic aromatic hydrocarbons PAH increases the risk of developing lung cancer. Human exposure is often demonstrated by increased internal levels of PAH metabolites and of markers for early biological effects, like DNA adducts and cytogenetic aberrations. Objective: This study aimed to assess whether the current exposure to PAH of coke oven workers in a Dutch plant induced biological effects, and to determine if these effects are influenced by tobacco smoking and by genetic polymorphisms for the glutathione S-transferase genes GSTM1 and GSTTI. Following correction for the smoking-related effects, no occupational induction of the effect biomarkers could be discerned. Also no significant interactions were observed between the various biomarkers. Conclusion: This study shows that in the examined plant, the occupational exposure to PAH does not result in measurable early biological effects. 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Sign in through your institution. Advanced Search. Search Menu. Article Navigation. Close mobile search navigation Article Navigation. Volume This article was originally published in. Journal Article. Box Oxford Academic. Google Scholar. Jeff G. Nico de Vogel. Truus C. Ton Schouten. Patricia Cramers. Lou Maas. Marcel H. Frederik-Jan van Schooten. Piet M. Hopmans Piet M. Cite Cite Joost H. Select Format Select format. Permissions Icon Permissions. Abstract Occupational exposure to polycyclic aromatic hydrocarbons PAH increases the risk of developing lung cancer. Issue Section:. You do not currently have access to this article. Download all slides. Sign in Get help with access. Institutional access Sign in through your institution Sign in through your institution. Get help with access Institutional access Access to content on Oxford Academic is often provided through institutional subscriptions and purchases. 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