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This website uses cookies, which may include third party cookies, to improve your browsing experience and in order to allow you to set your individual preferences for present and future use of the site. To find out more, read our Privacy Policy. The Medical City Department of Laboratory Medicine and Pathology operates under the discipline of laboratory medicine and provides relevant information for the diagnosis, prevention or treatment of disease, impairment of personal care, and assessment of health. The Laboratory, located at the Lower Ground Floor, is a fully equipped modern diagnostic facility offering a broad range of tests. The Laboratory processes over two million laboratory procedures annually. The Laboratory can also provide a vast majority of routine tests, as well as a variety of special procedures. Ninety nine percent of all testing is performed in-house. A home phlebotomy service is available for ambulatory and non-ambulatory patients. Contact Us Tel. Agustina Abelardo. Rose Lou Marie Agbay. Elizabeth Ann Alcazaren. Elizabeth Arcellana-Nuqui. Alejandro Arevalo. Jose Carnate, Jr. Gregorio, III Cortez. Sarah Jane Datay-Lim. Rouchelle Dela Cruz. Arvin Faundo. Maria Cecilia Lim. Pier Angeli Medina. Carmelita Navarro. Pedrito Tagayuna. Justine Alessandra Uy. About Us The Medical City Department of Laboratory Medicine and Pathology operates under the discipline of laboratory medicine and provides relevant information for the diagnosis, prevention or treatment of disease, impairment of personal care, and assessment of health. Therapeutic Application a. Cytapheresis a. Leukopheresis a. Platelet Reduction a. Erythrocytapheresis b. Donor Application a. Plateletpheresis C. Whole Blood B. Packed RBC C. Platelet Concentrates E. Fresh Frozen Plsama F. Cryoprecipitates G. Cryosupernates Blood typing A. Anti-HCV C. HBsAg D. Combination of Phadiatop, Total IgE c. Panel of 10 allergens Choices of specific individual allergens d. Crab f23 b. Shrimp f24 c. Squid f58 d. Blue mussel f37 e. Tuna f40 f. Fish cod f3 g. Salmon f41 Nuts a. Peanut f13 b. Almond f20 c. Cat dander e1 b. Dog dander e5 Insect a. Cockroach i6 b. Mosquito i7i Fruits a. Mango f91 b. Acacia t19 c. Bermuda grass g2 d. Johnson grass g10 e. Common ragweed w1 f. Common pigweed w14 Vegetables a. Tomato f25 Others a. Latex k82 Moulds a. Egg white f1 b. Egg yolk f75 c. Chicken meat f83 d. Pork f26 e. Beef f27 Dairy Products a. Cheddar cheese f81 b. Milk f2 Grains a. Wheat f4 b. Oat f7 c. Soya Bean f14 d. Barbiturates c. Benzodiazepines d. Cannabinoids Marijuana e. Cocaine f. Enterovirus B. Japanese B Encephalitis E. Varicella Zoster Virus Histopath Autopsy a. Complete - Including brain b. Limited - Brain not included Cell Block Cytology a. Large Specimen c. Medium Specimen d. Radical Resection e. Small-Diagnostic f. Small Specimen g. Grams Stain b. Grocotts c. PAS d. Reticulin e. Mucicarmine f. Ziehl Nielsen Acid Fast g. Elizabeth Y. Agustina D. Abelardo Dr. Elizabeth Ann S. Alcazaren Dr. Arcellana-Nuqui Dr. Jose M. Carnate, Jr. Gregorio B. Cortez III Dr. Sarah Jane L. Datay-Lim Dr. Rouchelle D. Pedrito Y. Tagayuna Forensic Pathology Dr. Raquel dR. Fortun Dr. Maria Cecilia F. Lim Hematopathology Dr. Alejandro E. Arevalo Nephropathology Dr. Alan T. Koa Neuropathology Dr. Edwin L. Munoz Orthopedic Pathology Dr. Carmelita V. Navarro Laboratory Quality and Safety Dr. Datay-Lim Ma. Lourdes L. Our Team. Agustina Abelardo Department of Medicine. Elizabeth Ann Alcazaren Department of Medicine. Elizabeth Arcellana-Nuqui Department of Medicine. Alejandro Arevalo Department of Medicine. Department of Medicine. Rouchelle Dela Cruz Department of Medicine. Arvin Faundo Department of Medicine. Alan Koa Department of Medicine. Maria Cecilia Lim Department of Medicine. Pier Angeli Medina Department of Medicine. Carmelita Navarro Department of Medicine. Pedrito Tagayuna Department of Medicine. Justine Alessandra Uy Department of Medicine. All rights reserved.

148728227 case-study-f

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Download now. More Related Content. Get Homework Done Homeworkping. This is our first time to encounter this kind of case and because of that, our group was interested in it. This case study will help the group in understanding the disease process of the patient. This would also help the group in identifying primary needs of the patient with Acute Pulmonary edema with severe preeclampsia. By identifying such needs and health problems arise the group can now formulate an individualized care plan for the patient that would address these needs and problems effectively. Effective management of the problems identified will help the patient to recover faster and maintain a holistic sense of wellness even while in the hospital. This case study would also equip the group with knowledge, skills and attitude on how to manage future patients with the same or similar disease. GLOBAL Cohort study - 62, consecutive pregnancies from , to describe the incidence, predisposing factors contributing to pulmonary edema in the pregnant patient. Fifty-one women 0. Most common causes: Tocolytics Aya et al. Address: Prk. History of present illness D. Past medical and Nursing History E. Personal, family and socio-economic history F. Hematocrit 0. Air is taken in via the upper airways the nasal cavity, pharynx and larynx through the lower airways trachea, primary bronchi and bronchial tree and into the small bronchioles and alveoli within the lung tissue. Move the pointer over the coloured regions of the diagram; the names will appear at the bottom of the screen The lungs are divided into lobes; The left lung is composed of the upper lobe, the lower lobe and the lingula a small remnant next to the apex of the heart , the right lung is composed of the upper, the middle and the lower lobes. Mechanics of Breathing To take a breath in, the external intercostal muscles contract, moving the ribcage up and out. The diaphragm moves down at the same time, creating negative pressure within the thorax. The lungs are held to the thoracic wall by thepleural membranes, and so expand outwards as well. This creates negative pressure within the lungs, and so air rushes in through the upper and lower airways. Expiration is mainly due to the natural elasticity of the lungs, which tend to collapse if they are not held against the thoracic wall. This is the mechanism behind lung collapse if there is air in the pleural space pneumothorax. Physiology of Gas Exchange. Each branch of the bronchial tree eventually sub- divides to form very narrow terminal bronchioles, which terminate in the alveoli. There are many millions of alveloi in each lung, and these are the areas responsible for gaseous exchange, presenting a massive surface area for exchange to occur over. Each alveolus is very closely associated with a network of capillaries containing deoxygenated blood from the pulmonary artery. The capillary and alveolar walls are very thin, allowing rapid exchange of gases by passive diffusion along concentration gradients. CO2 moves into the alveolus as the concentration is much lower in the alveolus than in the blood, and O2 moves out of the alveolus as the continuous flow of blood through the capillaries prevents saturation of the blood with O2 and allows maximal transfer across the membrane. Tightness and pain in the chest Wheezing, coughing Paleness Due to inadequate blood perfusion. Sweating Bluish nails and lips Due to inadequate blood perfusion. The cause of pulmonary edema in the presence of a hypertensive crisis is probably due to a combination of increased pressures in the right ventricle and pulmonary circulation and also increased systemic vascular resistance and left ventricle contractility increasing the hydrostatic pressure within the pulmonary capillaries leading to extravasation of fluid and edema. Acute pulmonary edema. N Engl J Med ; Pathophysiology Preeclampsia is a result of generalized vasospasm. The underlying cause of the vasospasm remains a mystery, although some of the pathophysiologic processes are known. In normal pregnancy, vascular volume and cardiac output increase significantly. Despite these increases, blood pressure does. This is probably because pregnant women develop resistance to the effects of vasoconstrictors, such as angiotensin II. In preeclampsia, however, peripheral vascular resistance increases because some women are sensitive to angiotensin II. They also may have a decrease in vasodilators. TXA2, produced by kidney and trophoblastic tissue, causes vasoconstriction and platelet aggregation clumping. PGI2, produced by placental tissue and endothelial cells, causes vasodilation and inhibits platelet aggregation. Vasospasm decreases the diameter of blood vessels, which results in endothelial cell damage and decreased EDRF. Vasoconstriction also results in impeded blood flow and elevated blood pressure. As a result, circulation to all body organs, including the kidneys, liver, brain, and placenta, is decreased. Blood urea nitrogen, creatinine, and uric acid levels begin to rise. This may result in edema and a reduction in intravascular volume, which causes increased viscosity of the blood and a rise in hematocrit. In response to reduced intravascular volume, additional angiotensin II and aldosterone trigger the retention of both sodium and water. Generalized edema may occur. This is manifested by elevation of liver enzymes in maternal serum. Dyspnea is the primary symptom. Blockage of lymphatic vessels Inability to remove excess fluid from interstitial space Accumulation of fluid in interstitial space Pulmonary Edema. Pulmonary edema is excess water in the lung. The normal lung contains very little water or fluid. It is kept dry by lymphatic drainage and a balance among capillary hydrostatic pressure, capillary oncotic pressure, and capillary permeability. Pulmonary edema result from obstruction of the lymphatic system. When lymph drainage is blocked, fluid accumulates in the lungs. Drainage can be blocked by an increase in systemic venous pressure, which elevates the hydrostatic pressure of the large pulmonary veins into which the pulmonary lymphatic system drains. Drainage also can be obstructed by compression of lymphatic vessels by edema, tumors, and fibrotic tissue. Unya cge pa jud kog ka lipong. Mao nang paminaw nako laing jud kaayo ako lawas. Assist client in performing ADL Place the client in trendelenbur g position. Maintain adequate ventilation. Instruct client to sit and dangle the feet before standing. Advise client To promote safety To promote venous return To promote oxygenatio n and good blood circulation To prevent orthostatic hypotensio n After 4 hours of nursing interventio n, the client will exhibit decrease in oxygen demand and ability to conserve energy. Iron and folate are necessary for red blood cell production. You may urinate more often when you take this medicine. This may decrease the pressure in your blood vessels and improve your symptoms. Tell him if you are allergic to any medicine. Keep a list of the medicines, vitamins, and herbs you take. Include the amounts, and when and why you take them. Bring the list or the pill bottles to follow-up visits. Carry your medicine list with you in case of an emergency. Follow up with your primary healthcare provider or pulmonologist in 7 to 10 days or as directed. Write down your questions so you remember to ask them during your visits. Manage pulmonary edema. Too much liquid can increase your risk for fluid build up. Weigh yourself at the same time every morning after you urinate, but before you eat. Weight gain can be a sign of extra fluid in your body. Return to activities slowly, and do more each day. You may have trouble breathing when you are lying down. Use foam wedges or elevate the head of your bed. This may help you breathe easier while you are resting or sleeping. Use a device that will tilt your whole body, or bend your body at the waist. The device should not bend your body at the upper back or neck. Alcohol can worsen your symptoms and increase your blood pressure. If you have heart failure, alcohol can make it worse. Do not take street drugs, such as cocaine. Smoking and drugs can make your condition and symptoms worse. Ask for information if you need help quitting. Ask your primary healthcare provider about the symptoms of high altitude pulmonary edema HAPE. Ask what to do if you get these symptoms. Wrap IV infusion solution in foil or opaque material to protect from light Monitor for possible adverse effects, such as apprehension, restlessness, retrosternal pressure, palpitations, diaphoresis, abdominal pain Furosemide Lasix Diuretic action, inhibiting the reabsorption of sodium and chloride from the ascending loop of Henle Pulmonary edema Administer via slow IV bolus at a dose of 10—40 mg over 1—2 minutes Monitor urine output hourly Assess for possible adverse effects such as dizziness, vertigo, orthostatic hypotension, anorexia, vomiting, electrolyte imbalances, muscle cramps, and muscle spasms. It also helped us understood the causes and effects of the diseases that enabled us to determine the predisposing and precipitating factors and traced the pathophysiology of these disorders. This also had given us the knowledge to identify where and when it had started and how the disease progressed and we had also interpreted the laboratory and diagnostic exam results of the client and recognized the implication of it. We also identified the different pharmacologic treatments indicated to the condition, considering the effects, actions and different nursing considerations with regards to the. We have also identified and formulated the nursing interventions that we could render to the patient that will help us attain our goal of care to our patient basing from the nursing care plan we have formulated. Furthermore, vital signs are expected to stabilize. Recommendation On the basis of the findings of this study, the following measures are recommended: 1. Client should take his prescribed medications religiously. He must create a schedule in order for him to be guided as when to take the medicines and for him not to be able to forget in doing so. Follow the prescribed diet. His prescribed diet is a low-salt, low-fat diet, therefore client should avoid salty and fatty foods and client must take note that all canned goods are high in sodium even if it says that it is good for the heart. Have an oral fluid intake with in cardiac tolerance. Lifestyle modification is also important in order to prevent the severity of the condition that will further contribute complications such as cessation of smoking and drinking alcoholic beverages. Visit his doctor regularly for constant check-ups and to continuously monitor his condition. Evaluation of the objectives of the study After few days of conducting study about the case of lorna, we were able to trace the history of her disease locally, nationally and globally. By understanding fully the mechanism and effects of the disease to the patient, we have interpreted different laboratory results related to her condition. Appropriate therapeutic care was well planned and provided to the client.

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