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Our brain interacts with other tissues, which can regulate and alter the brain's functions. Recent studies reveal that changes in the environment around the brain such as blood vessels and guts are closely linked to the onset and progression of Alzheimer's disease. In this symposium, we aim to discuss the latest research findings from distinguished speakers and explore how these discoveries could lead to the development of effective treatments. Brain and body communicate and work together to maintain homeostasis, which is the state of equilibrium or balance in the internal environment of our body. In this symposium, there will be cutting-edge researchers on bi-directional communication between brain and peripheral organs. This symposium is dedicated to improving our understanding of transient oscillations in the beta and gamma frequency bands, which are increasingly recognised as crucial for interpreting the neural mechanisms of cognition and behaviour. The aim is to further explore the underlying neural mechanisms associated with these cognitive processes and to identify new avenues of research. The agenda includes an examination of the role of beta bursts in predictive coding, the interplay between beta and gamma bursts in cognitive functioning, the neural circuitry underlying beta and gamma bursts, and the clinical implications that these bursts may have. It is expected that the symposium will act as a conduit for the exchange of the latest research on beta and gamma bursts and their role in higher-level cognitive processing, encompassing studies in mice, monkeys and humans. Humans and many other organisms use their five senses to gather the physicial and chemical information from the external world. The noceiceptive system and its associated touch, itch and pain sensations rely not only on the external senses, but require interceptive sensing. This symposium will explore recent advances in our understanding of mammalian somatosensation that highlight the use of cutting-edge technologies and reveal novel mechanisms involved genes, neural circuits and behavior. We will also reserve time for a short talk that represents another type of interoceptive sensing in animal. Our goal is to encourage the sharing unpublished data, promote lively discussions and foster new collaborations across the field. Communication, crucial for human interaction, encompasses two key aspects: perception and production. This symposium delves into the advanced Brain-Computer Interface BCI technology, focusing on neural mechanisms underlying these processes and state-of-the-art deep learning architectures. This symposium deals with crucial challenges such as intelligibility, controllability based on speech processing in human brain. We expect this symposium will provide insights into innovative frameworks and applications revolutionizing the field of speech BCI. The neuroimmune interaction emerges as a key player in regulating the physiology and pathology of the brain as well as controlling immune functions. The crosstalk between the nervous system and the immune system embraces multiple types of organs, tissues, and cells, requiring comprehensive investigation at multiple levels. To facilitate understanding of current advances in neuroimmune interaction, we aim to collect leading scientists focusing on each critical player - traditional immune system, brain-blood barrier, glial cells, and even brain-resident immune cells. This session will provide a comprehensive introduction and discussion of the diverse types of neuroimmune interaction and their physiological and pathological consequences, with the hope of incubating international collaboration. In the past decade, human pluripotent stem cell derived 3D neural organoids have become invaluable tools for modeling early developmental processes and central nervous system disorders. As neuropsychiatric and neurodegenerative disorders are shaped by underlying complex human genetic backgrounds which ultimately modify the disease risk, onset, and progression, human induced pluripotent stem cells hiPSCs grants direct access to unique human genetic backgrounds for experimental exploration. This symposium aims to showcase ongoing research by early-stage investigators who have utilized 3D neural organoid models to gain insights into the complexities of brain disorders and neurodevelopment. By focusing on the intersection of advanced cellular models and complex genetic influences, the speakers will present recent findings contributing to our understanding of translational research. The critical role of the hippocampus in spatial and episodic memory has been documented across many species across the phylogenetic tree. While the field has benefited immensely from the wide range of studies on hippocampal function, it still remains a challenge to bring together unique insights from each species into a comprehensive understanding of how episodic memory works. In this symposium, expert neuroscientists in the field of memory research across rodents, non-human primates, and humans will share their recent findings on hippocampal processes measured at the level of single neurons, brain oscillations, and functional neuroimaging. In discussing both common and unique characteristics of spatial and episodic memory across multiple species, we hope to provide a fresh perspective on how such evolutionarily ancient neural mechanisms give rise to our daily experience of episodic memory. By bridging research across subfields of behavioral and cognitive neuroscience, this symposium will interest researchers from various areas of neuroscience and the broad KSBNS community. Astrocytes play multiple and essential roles in brain function. In response to various pathological stimuli, reactive astrocytes exhibit molecular, structural and functional alterations and play an important role in the pathogenesis of neurological disorders. Although in vivo research ranging from molecular and cellular levels to behavioral levels are challenging, research into the relevant mechanisms and diagnosis of neurological disorders through reactive astrocytes is actively ongoing. In this symposium, four international speakers will share their recent findings that provide new insights into RBP functions in neurons. Kwok-On Lai City University of Hong Kong will discuss a strategy that involves transplantation of human induced pluripotent stem cells hiPSC to demonstrate how chronic behavioural stress interacts with ALS-related FUS gene mutation to aggravate the disease phenotypes of human neurons in vivo. Mechanobiology in the nervous system explores how mechanical forces influence the structure, function, and development of neural tissues. Experts will discuss topics such as mechanotransduction primarily mediated by mechanosensitive ion channels in brain cells and the impact of biomechanical forces on signal transduction and cellular homeostasis. Cutting-edge research will unravel the fascinating interplay between mechanics and neurobiology and broaden our understanding of the complexity and resilience of the nervous system. Despite the paramount importance of adverse experience during early development on cognition and emotion in later stages of life, only a small number of studies have addressed this issue. In this symposium, four distinguished researchers from Hong Kong, Australia, and Korea will introduce the latest findings from their laboratories using rodents and human subjects. The audience in this symposium session is guaranteed to gain valuable insights into how stress and trauma during different stages of development modulate neurobehavioral systems in adolescence and adulthood. In this symposium, we bring together international speakers who are investigating novel aspects of RNA biology, in the context of neurodevelopment, experience-dependent plasticity, and other aspects of brain function, and dysfunction. This symposium will share novel insights regarding the various ways in which RNA biology can mediate, and modulate, a wide range of processes within the developing and mature nervous system. Symposium attendees will be encouraged to consider RNA not only as medium for information transfer between DNA and protein levels, but also the wider realm of functional RNAs, and their key roles in brain development, function, and dysfunction. Over the past ten years, there has been a significant surge in interest in the lateral habenula LHb due to recent discoveries highlighting its crucial role in controlling negatively driven behavior and its involvement in reward prediction as well as stress response. We will discuss how emotional contagion promotes coping behaviors in conditions of heightened stress, and how this part of the emotional brain can determine social behaviors. We will expand the discussion to the role of the lateral habenula during the rewarding and aversive conditions in virtual reality as well as during conditioned inhibition, a fundamental component of associative learning. This symposium will shed light on the recent studies regarding the more complex function of the LHb. Repeated exposure to a drug of abuse induces neuronal and synaptic changes in the brain, leading to the compulsive seeking and use of drugs, and the loss of control over drug consumption. This addiction imposes a significant social and economic burden on the community. However, the processes through which addiction memory is established, perpetuated, and amplified remain unclear. Research on addiction memory utilizes various tools and concepts to elucidate the causal components of addictive states, understand the neural mechanisms involved, and develop effective therapeutic interventions. At this symposium, prominent researchers actively investigating addiction memory will present new findings that contribute to a deeper understanding of how molecular, cellular, and circuit changes in the nervous system dictate addiction memory and compulsive behavior. Proper synaptic plasticity is essential for neural circuit function, and conversely, altered synaptic dynamics impair neuronal activity and are thought to underlie the pathology of brain disorders including ASDs. This idea is supported by the observation of increased synapse number in the frontal lobe in ASD brains. The prefrontal cortex PFC is a brain region associated with higher cognition that is shown to be disrupted in ASDs; yet, key questions remain unanswered concerning the neurobiological mechanisms that mediate the detrimental effects of altered synaptic and neuronal activity on PFC function. This symposium will bring together rising neuroscientists, leading experts in the synaptic and cellular basis of neuronal plasticity, to discuss the most recent advances in the field. A diverse group of 4 speakers will present recent findings on PFC function and dysfunction that are relevant to the major theme of this symposium. With the aging society, there is an increasing societal demand for research in the field of neuroscience, particularly in degenerative brain diseases. We would like to introduce female scientists actively contributing to various research methods in the diverse field of neurodegenerative diseases to KSBNS participants. Through this, we hope to encourage the influx of more women scientists into the field of neuroscience, introduce new perspectives in neurodegenerative disease research, and foster numerous collaborations. This interdisciplinary symposium brings together experts in primate systems neuroscience and medical research. It explores primate intelligence's complexity and its implications for understanding the human brain, particularly in the context of brain diseases. Topics include natural intelligence in social settings, neural circuit mechanisms for efficient decision-making, and cutting-edge genetic techniques for modeling mental diseases in non-human primates. The symposium welcomes researchers, clinicians, students, and anyone interested in primate research, systems neuroscience, and brain disease studies. It offers a comprehensive overview of current research and fosters discussions on future directions in this field. The 'Emerging Insights in Neuroimmunology and Pain' symposium explores the nexus between the nervous system and immune responses in pain. Focusing on how interleukins, microglia, and macrophages affect neuropathic pain, it integrates cutting-edge research on molecular pathways with clinical implications. Highlights include gender-specific neuroimmunological responses and novel therapeutic strategies. This interdisciplinary event appeals to researchers and clinicians, fostering dialogues and inspiring new treatments. Associate Professor Cheung will then follow with research showing the novel link between vascular injury and neuroinflammation, showing a potential causal pathomechanism of cognitive dysfunction associated with brain hypoxia due to vascular injury. This research offers new therapeutic targets to treat neurovascular conditions such as vascular dementia. Dr Senesi will then propose how the misfolding of proteins such as prion in Creutzfeldt-Jakob disease is a key mechanism that may apply across many neurological disorders including dementia. Such novel theory allows new therapeutic avenues to interfere with protein misfolding such as antibodies and antisense nucleotides to treat various neurological disorders. Professor Jee Hyun Kim will chair with Assistant Professor Ikenaka co-chair to lead an impartial discussion of the presented findings and potential questions. Brain development is a highly regulated phenomenon in which a variety of processes are involved, including synaptic plasticity, synapse formation and elimination, excitation-inhibition balance, transcriptional control, and translational control. This symposium aims to capture some of the latest findings in neural development and its malfunction that is implicated in disease states like epilepsy. We have brought together four internationally recognized speakers with wide-ranging research interests and expertise. He will speak about how a limbic region, anterior piriform olfactory cortex is involved in temporal lobe epilepsy. Zhuo HUANG Peking University will speak about epilepsy and how environmental cues can trigger seizure bouts in a phenomenon called conditioned seizure memory. Jacque IP The Chinese University of Hong Kong will speak about visual cortical development and its relationship with an epilepsy-related neurodevelopmental disorder. Together, the topics and methodology brought forth will illustrate our current understanding and innovation in neural development and epilepsy, making this symposium a timely and engaging one. Neurodegenerative brain disorders present complex challenges that extend beyond the confines of the brain itself. Recent research suggests that the pathogenesis of neurodegenerative disorders is not solely confined to localized brain dysfunction but involves a dynamic interplay with various extra-cranial factors. This symposium aims to illuminate the intricate web of connections between the brain and external organs, with a special emphasis on the gut, immune system, and microbiome. Understanding the mechanisms operating both within the brain and in collaboration with peripheral organs is crucial for advancing our comprehension of these debilitating conditions. By delving into the molecular and cellular mechanisms within the brain and exploring the interconnectedness with peripheral organs, we aim to unravel the complex tapestry of neurodegenerative disorders. In the session, we will explore the synergy of electrophysiology, calcium imaging and MRI in unraveling the mysteries of perception and short-term memory. This interdisciplinary approach will offer a comprehensive view of how senses are interpreted, stored, and manipulated. The session will foster collaboration among the scientists in the fields of cognitive, systems, and molecular neuroscience. Sensory information is processed in a dynamically regulated manner to successfully adapt to an ever-changing environment. During the session we will explore the dynamic network changes in response to rule-change, updated reward information, or simply during errors in short-term memory. We will explore the synergy of electrophysiology, calcium imaging and MRI in unraveling the mysteries of perception and short-term memory. Our quest to understand memory builds on fundamental work addressing the molecular, synaptic, physiological and behavioral processes engaged during learning. Over the last decade rapid technological advancements on all these level, harnessing progress in machine learning, high-density imaging and recording, high-throughput genomics and proteomics and genetic tools for circuit characterization and manipulation, has accelerated progress in the field and uncovered new and surprising connections between levels of investigation. This session will showcase the application of these emerging approaches, including large-scale in vivo electrophysiology, whole-brain synaptome mapping, and virtual reality based brain-machine interfaces, to long-standing questions of how mammalian brains encode and use memories, as well as inspire researchers across all fields to harness these tools in their own work. The suggested symposium theme, 'Integrative Insights into Neurobiology: From Physiological Neural Models to Neurodegenerative Diseases and Drug Discovery,' encompasses a comprehensive exploration of various facets of neuroscience research. The first topic emphasizes the use of physiological neural model systems to delve into studies related to brain development, neuronal maturation, and neuroinflammation. The second topic shifts the focus to utilizing microplate readers in unraveling neurodegenerative diseases, specifically highlighting prion research and protein aggregation. Finally, the third topic underscores the power of in vitro systems using human induced pluripotent stem cell iPSC models for advancing drug discovery. This amalgamation of diverse themes aims to encompass the broad spectrum of neuroscience while emphasizing practical and applicable research domains. The symposium unveils novel perspectives on information storage and utilization, framing them as highly dynamic information processing phenomena. The plasticity of neural networks enables long-term stored information to enhance cognitive abilities, and the decision-making process intricately integrates current sensory input with stored value information. The symposium delves into the intricate information integration processes of cognition at the neural network level and aims to shed light on how these dynamic cognitive processes are represented and stored in the brain. Topics include the influence of neural activity during sleep on memory formation, the role of neuroplasticity in enhancing cognitive abilities, and the complex neural integration of olfactory and auditory input in decision-making. Through the symposium, attendees will gain insights into the cutting-edge research conducted by emerging neuroscientists who are actively investigating the neural correlates of cognition. Autism spectrum disorder ASD is a neurodevelopmental disorder marked by deficits in reciprocal social communication and the presence of restricted and repetitive patterns of behavior. It is highly heritable but regarded as a spectrum disorder, co-occurring with other diverse conditions. This symposium aims to present the most recent advancements in understanding neural circuit mechanisms governing pathological conditions in ASD. Esteemed neuroscientists will be featured in this symposium, encompassing both male and female speakers from the United States, Japan, and Korea. Each presenter in this symposium has employed state-of-the-art methodologies to delineate critical neural circuits underlying social deficits, sleep disturbances, memory deficits, and sensory dysfunctions in ASD mouse models. The symposium anticipates drawing a considerable audience interested in new aspects of brain dysfunctions in ASD. Neurodevelopmental disorders are types of disorder that alters neurological development, causing difficulties in a wide range of brain functions. Children with neurodevelopmental disorders can experience difficulties with language and speech, motor skills, behavior, memory, learning, or other neurological functions. Causes are heterogeneous ranging from social deprivation, genetic and metabolic diseases, immune disorders, infectious diseases, nutritional factors, physical trauma, and toxic and environmental factors. However, our current knowledge of neurobiological mechanisms underlying neurodevelopmental disorders is far from complete. The current symposium aims to bring attention to research on neurobiological mechanisms of neurodevelopmental disorders. The concept of 'brain plasticity' often conjures images of synaptic connections between neurons, both in the minds of neuroscientists and the general public. Our goal is to expand beyond this traditional viewpoint by proposing a symposium focused on the plasticity of astrocytes surrounding synapses. This session aims to highlight the significance of morphological plasticity—termed here as morphoplasticity—and explore its role in influencing cognitive functions within the brain. Although modern neuroscience reveals the mechanism of specific brain functions and recent artificial neural network models demonstrate powerful performance on specific tasks, understanding how neural networks whether implemented naturally in the brain or artificially could generate such functions have been considered still enigmatic. Thus, this symposium session will focus on the effort to find out the general principles underlying how neural networks work in computational neuroscience. The topics in this symposium are selected and organized to promote in-depth discussions from a variety of perspectives, ranging from the conventional dynamics of neurons and synapses to the recently highlighted properties of neural networks, such as modulation or information processing cost. The purpose of these Terms and Conditions is to stipulate rights, obligations, and responsibilities of users and the Korean Society for Brain and Neural Sciences and other necessary matters while using the integrated member service hereinafter, referred to as 'Service' provided by the Korean Society for Brain and Neural Sciences hereinafter, referred to as 'The Korean Society for Brain and Neural Sciences' homepage. 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Williams University of Texas Southwestern Medical Center Effects of metabolic state on the regulation of melanocortin neurons Min-Seon Kim University of Ulsan Regulation of skeletal muscle mass and strength by hypothalamic neurons. Time table Speaker Affiliation Title Inah Lee Seoul National University Hippocampal correlates of contextual object recognition memory in nonhuman primates Sebastien Royer Korea Institute of Science and Technology Memory trace of a butterfly encounter Joshua Jacobs Columbia University Probing the neural basis of human memory with direct recordings of place and grid cells and traveling waves Sang Ah Lee Seoul National University Constructing memories across space and time in human episodic memory. Time table Speaker Affiliation Title Ying-Shing Chan The University of Hong Kong Balance of excitatory-inhibitory transmission in the developing vestibular nucleus tunes the brain circuitry for reflexes and spatial navigation Jee Hyun Kim Deakin University Chronic infancy or adolescent stress sex-dependently alters hypothalamic-pituitary-adrenal axis and extinction of conditioned fear June-Seek Choi Korea University Differential brain responses to chasing stress in the adolescent and adult brain Ji-Won Hur Korea University Neural correlates of vulnerability to nonsuicidal self-injury in response to childhood adversity. Time table Speaker Affiliation Title Manuel Mameli The University of Lausanne Serotonin plasticity in habenula during emotional contagion protects from negative affective states ChiHye Chung Konkuk University Sociality regulation by cortico-habenula pathway under the stress HyungGoo Kim Sungkyunkwan University Diversity of lateral habenula neurons in reward and aversive processing Jung-soo Han Konkuk University The role of the lateral Habenula in conditioned inhibition. Kim Seoul National University Natural intelligence enables efficient information processing: extracting value from tactile and visual inputs facilitates effficiency in primates Soo Hyun Park Korea Advanced Institute of Science and Technology Studying dynamic visual processing in non-human primate model Youngjeon Lee Korea Research Institute of Bioscience and Biotechnology Research on brain disease models using non-human primates. Time table Speaker Affiliation Title Chihiro Tohda University of Toyama From basic study to clinical study for realizing new botanical medicines Christine Cheung Nanyang Technological University Chronic cerebral hypoperfusion induces endothelial dedifferentiation that leads to pathological venous remodeling Matteo Senesi University of Melbourne Prions as prototypical agents for neurodegenerative diseases: mechanisms, therapeutics and beyond Kensuke Ikenaka Osaka University Exploring treatments for Parkinson's disease through understanding the mechanisms of alpha-synuclein aggregation and propagation. A powerful in vitro system using human iPSC models enables facilitating drug discovery. Schneider New York University Cortical error detection enables skilled acoustic behaviors Jaekyung Kim Korea Advanced Institute of Science and Technology Exploring circuit dynamics during sleep and neural representations in motor learning Ain Chung Korea Advanced Institute of Science and Technology Hippocampal circuit mechanisms underlying social information processing enhancement. Time table Speaker Affiliation Title Mark van Rossum University of Nottingham Energy efficient learning in biological and artificial neural networks Louis Kang RIKEN Computational demands can influence seizure susceptibility in recurrent neural networks Hyojin Bae Seoul National University A normative principle governing memory transfer in cerebellar motor learning Taegon Kim Korea Institute of Science and Technology Dependence of functional diversification by modulatory neurotransmitters on the architecture of neural networks. Terms of Use. 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If the data provided by the member is found to be false If it involves a criminal activity When planning to use the Service or implementing it for the purpose of undermining national interests or social and public interests In case of stealing someone else's service ID and password In case of damaging other's reputation or disadvantaging others In case of impairing the healthy use of the Service, such as harming the Service In case of violation of other relevant laws or the conditions of use set by the Korean Society for Brain and Neural Sciences. The Korean Society for Brain and Neural Sciences shall be exempted from responsibility for failing to provide the Service if this failure is caused by natural disasters or the equivalent force majeure. The Korean Society for Brain and Neural Sciences shall not be responsible for any service use impediments caused by reasons attributable to the member. The Korean Society for Brain and Neural Sciences shall not be held liable for expecting benefits by the member's using the Service or damages from the materials obtained through the Service. The Korean Society for Brain and Neural Sciences shall not be responsible for reliability, accuracy, etc. If there is a dispute between The Korean Society for Brain and Neural Sciences and its member in connection with the use of the Service, the Korean Society for Brain and Neural Sciences and the member shall make all necessary efforts to smoothly resolve the dispute. Notwithstanding the provision of paragraph 1, if a lawsuit is filed against a dispute arising from the use of the Service, the court governing the location of the Korean Society for Brain and Neural Sciences shall be the competent court. Privacy Policy. Items of personal information collected and methods of collection The Korean Society for Brain and Neural Sciences collects the following personal information for customer center and customer consultation. Name, business name, e-mail, phone, fax, login ID, password or the following information that may be generated and collected during the use of the service or the processing of the business Service usage record, access log, access IP information, cookie 2. Purposes of collecting and using personal information The Korean Society for Brain and Neural Sciences collects personal information in the following methods. Customer Center, customer consultation bulletin board The Korean Society for Brain and Neural Sciences uses the collected personal information for the following purposes. Handling and reporting of service requests, answers to customers' questions Use for marketing and advertising Delivering new service product development information in the customer consultation 3. Providing and sharing personal information In principle, the Korean Society for Brain and Neural Sciences does not provide the users' personal information outside. In accordance with regulations of laws or in case there is a request by the investigation agency for the purpose of investigation in accordance with the procedures and methods prescribed in laws 4. Consignment of handling of collected personal information The Korean Society for Brain and Neural Sciences does not consign the handling of the customer' personal information to an outside company without the customer's consent. Retention and use period of personal information In principle, after the purpose of collecting and using personal information is achieved, the corresponding information is destroyed without delay. Login ID, password, business name, name, e-mail, phone, fax Reasons for retention: handling and reporting of the work requested by customer and sending an operation report Period of retention: when destroying the operation contract 6. Procedure and method of destroying personal information In principle, the Korean Society for Brain and Neural Sciences destroys the corresponding information without delay after achieving the purpose of collecting and using personal information. Procedure of destruction After the purpose is achieved, the information entered by the member for membership is destroyed after being stored for a certain period of time according to the reasons for protecting the information see Period of Retention and Use under internal policies and other relevant laws. This personal information shall not be used for any purpose other than to be retained unless in accordance with the law. Methods of Destruction The personal information printed on paper shall be shredded using a shredder or incinerated The personal information stored in an electronic file shall be deleted by using a technical method that does not allow restoring the records. Rights of user and legal representative and how to exercise them Users and legal representatives may at any time view or modify the registered personal information of themselves or their children under the age of 14 and request termination of the membership. Matters concerning the installation, operation and refusal of The Korean Society for Brain and Neural Sciences installs and operates a device that automatically collects personal information such as 'cookie' or 'session' that frequently finds and stores your information. The Korean Society for Brain and Neural Sciences uses cookies for the following purposes Purposes of using cookies, etc. How to refuse cookies Example: As ways to refuse cookies, by selecting options in your web browser, you can allow all cookies, check them each time they are stored or refuse to save any cookies. Person in charge of personal information management The Korean Society for Brain and Neural Sciences designates a department in charge of personal information management as follows to protect customers' personal information and handle complaints concerning personal information. Refund Policy. Modeling the interactions of diet and genetics with in vitro models of the human brain. Theta long-range synchrony in the cerebral cortex in support of perception and action. Clear optically matched panoramic access channel technique COMPACT for large-volume deep brain imaging to study nerve injury and inflammatio. Spatiotemporal electrocorticographic patterns during brain stimulation-induced auditory perception. Exploring mmune-microbiome-brain axis in a neurodevelopmental disorder mouse model. Stress induces behavioral abnormalities by increasing expression of phagocytic receptor MERTK in astrocytes to promote synapse phagocytosis. Pathological roles of blood brain barrier dysfunction in the development of neurodegenerative diseases. Unveiling disease-specific impacts of NRXN1 deletions on cortical synaptic function and plasticity using human iPSC-derived neural cells. Early cellular changes in neurons linked to presymptomatic Alzheimer's disease. Hippocampal correlates of contextual object recognition memory in nonhuman primates. Probing the neural basis of human memory with direct recordings of place and grid cells and traveling waves. Tonic excitation by astrocytic GABA causes neuropathic pain through augmenting neuronal activity and glucose metabolism. Synapse-enriched long non-coding RNAs regulates fear memory formation involving dendritic protein synthesis. Carbon nanotube platform regulating intracellular calcium and physiological functions of astrocytes differentially across brain regions. Balance of excitatory-inhibitory transmission in the developing vestibular nucleus tunes the brain circuitry for reflexes and spatial navigation. Chronic infancy or adolescent stress sex-dependently alters hypothalamic-pituitary-adrenal axis and extinction of conditioned fear. Differential brain responses to chasing stress in the adolescent and adult brain. Neural correlates of vulnerability to nonsuicidal self-injury in response to childhood adversity. Serotonin plasticity in habenula during emotional contagion protects from negative affective states. Linking neuronal activity in the prefrontal cortex to ASD symptoms: a reverse-translational assessment in ASD model mice. Microglia-mediated noradrenergic gating of spine enlargement and learning in the medial prefrontal cortex. Synapse-level mechanisms of serotonin actions on neuronal plasticity in the prefrontal cortex. Markerless motion capture system for social behavior analysis in normal and disease model primates. Natural intelligence enables efficient information processing: extracting value from tactile and visual inputs facilitates effficiency in primates. From viral infection to neuropathic pain: a closer look at STING activation in nociceptors and microglia. Spinal interleukin contributes to neuropathic pain after peripheral nerve injury through interleukin receptor2 in mice. Chronic cerebral hypoperfusion induces endothelial dedifferentiation that leads to pathological venous remodeling. Prions as prototypical agents for neurodegenerative diseases: mechanisms, therapeutics and beyond. Exploring treatments for Parkinson's disease through understanding the mechanisms of alpha-synuclein aggregation and propagation. Suppression of piriform cortex alters brain-wide dynamics and alleviates seizures in temporal lobe epilepsy. Cortical visual impairment: unraveling insights into neurodevelopment and epilepsy-related disorders. Termination of convulsion seizures by destabilizing and perturbing seizure memory engrams. Comparative genomics upon neuronal stimulation delineates neuronal regulatory elements linked to neuropsychiatric traits including epilepsy. Colonization of non-cortical brain regions by monocyte-derived macrophages with a Dnmt3a mutation causes motor pathology. Thalamocortical networks dynamically encode statistical properties of sensory stimuli to enable perceptual inference. Distributed encoding and opposite directional activities in the PPC during errors in short-term memory. Spatially specific reward information in visual areas during closed-loop naturalistic interaction. Neuropixels 2. Volitional activation of remote place representations with a hippocampal brain—machine interface. Using physiological neural model systems to study neurodevelopment, neuronal maturation and neuroinflammation. Uncovering neurodegenerative diseases using microplate readers: prion research and protein aggregation. Exploring circuit dynamics during sleep and neural representations in motor learning. Hippocampal circuit mechanisms underlying social information processing enhancement. Circuit mechanism underlying fragmented sleep and memory deficits in 16p Perturbed cell fate decision by a schizophrenia-associated AS3MT isoform during corticogenesis. Deciphering mechanisms of perinatal white matter injury-induced neuropsychiatric outcome. Astroglial Hmgb1 regulates postnatal astrocyte morphogenesis and cerebrovascular maturation. Collaborations among peers: astrocyte-neuron interactions in plasticity and brain disorders. Computational demands can influence seizure susceptibility in recurrent neural networks. Dependence of functional diversification by modulatory neurotransmitters on the architecture of neural networks.

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