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Always start with lower doses due to differences between individual body weight, tolerance, metabolism, and personal sensitivity. See responsible use section. It is not a recommendation and should be verified with other sources for accuracy. Early discussion over DCK has revolved around speculation over claims of antibacterial or immunosuppressant properties. DCK has recently become easily accessible through online research chemical vendors \\\\\\\\\\\\\[2 \\\\\\\\\\\\\] where it is being sold as a designer drug. Very little data exists about the pharmacological properties, metabolism, and toxicity of DCK, and it has a very brief history of human usage. It is strongly recommended that one use harm reduction practices if choosing to use this substance. Deschloroketamine, or 2-Phenyl methylamino cyclohexanone, is classed as an arylcyclohexylamine drug. Ayrlcyclohexylamine drugs are named for their structures which include a cyclohexane ring bound to an aromatic ring along with an amine group. Descholoroketamine contains a phenyl ring bonded to a cyclohexane ring substituted with an oxo group cyclohexanone. An amino methyl chain -N-CH 3 is bound to the adjacent alpha carbon R 2 of the cyclohexanone ring. Descholoroketamine is a chiral molecule and is often produced as a racemate. Des- is a prefix used in chemistry to denote the absence of a functional group in this case 'chloro' hence deschloroketamine is named for lacking a chlorine substitution on its phenyl ring, which is found in ketamine. Due to the lack of research regarding the substance, all discussion regarding the pharmacology of it is purely based on its structure and subjective effect similarities to other arylcyclohexylamine dissociatives such as 3-MeO-PCP , PCP and MXE. NMDA receptors, a type of glutamate receptor, allow for excitatory electrical signals to pass between neurons in the brain and spinal column; for the signals to pass, the receptor must be open. Dissociatives inactivate the NMDA receptors by blocking them. It has therefore come to be considered a more suitable ketamine substitute than many other popular arylcyclohexylamines currently available on the research chemicals market, although users are advised to exercise extreme caution due to the health risks it may pose if it does possess its speculated antibacterial properties. The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects should be taken with a grain of salt and will rarely if ever occur all at once, but heavier doses will increase the chances and are more likely to induce a full range of effects. Likewise, adverse effects become much more likely on higher doses and may include injury or death. There are currently anecdotal reports which describe the effects of this compound within our experience index. The toxicity and long-term health effects of recreational DCK use do not seem to have been studied in any scientific context and the exact toxic dosage is unknown. This is because DCK has very little history of human usage. Anecdotal evidence from people who have tried DCK within the community suggests that there do not seem to be any negative health effects attributed to simply trying this drug at low to moderate doses by itself and using it sparingly but nothing can be completely guaranteed. Independent research should always be done to ensure that a combination of two or more substances is safe before consumption. It is strongly recommended that one use harm reduction practices when using this substance. As with other NMDA receptor antagonists, the chronic use of DCK can be considered moderately addictive with a high potential for abuse and is capable of causing psychological dependence among certain users. When addiction has developed, cravings and withdrawal effects may occur if a person suddenly stops their usage. Tolerance to many of the effects of DCK develops with prolonged and repeated use. This results in users having to administer increasingly large doses to achieve the same effects. After that, it takes about 3 - 7 days for the tolerance to be reduced to half and 1 - 2 weeks to be back at baseline in the absence of further consumption. DCK presents cross-tolerance with all dissociatives , meaning that after the consumption of DCK all dissociatives will have a reduced effect. In terms of its long-term health effects when used repeatedly and with excess for extended periods of time, DCK seems to exhibit almost identical bladder and urinary tract problems to those found within ketamine but to a lesser extent. This is suspected to be because DCK is more potent than ketamine, meaning that less of the drug needs to be consumed to produce analogous effects. Symptoms of ketamine-induced cystitis can become extremely serious and include:. Although many psychoactive substances are safe on their own, they can become dangerous and even life-threatening when combined with other substances. The list below contains some common potentially dangerous combinations, but may not include all of them. Certain combinations may be safe in low doses of each but still increase the potential risk of death. As such, it may contain incomplete or wrong information. You can help by expanding it. Deschloroketamine Deschloroketamine Molecular structure of Deschloroketamine. Retrieved from ' https: All articles with unsourced statements Articles with unsourced statements Psychoactive substance Research chemical Dissociative. Navigation menu Personal tools English Create account Log in. Views Read View source View history. Community Network Youtube Good vibes. This legality section is a stub.

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