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Download scientific diagram | Depletion of Top2 during S Phase Causes H2ASP Enrichment at Nicola Minchell; Andrea Keszthelyi; Jonathan Baxter.
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To define potential biomarkers for TOP2 poisons, CRISPR hits were overlapped Ceccaldi R, Sarangi P, and D'Andrea AD, The Fanconi anaemia.
Andrea Banfi. Department of Physics and Astronomy, University of Sussex, finitely heavy top2). In fact, only by taking into account N3LO corrections is.
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Victor Gonzalez-Huici, Daniele Fachinetti, Andrea Cocito, Gioacchino Top2 and Rpb3 signal distribution in S-phase along the entire chromosome III.
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Tyrosyl-DNA phosphodiesterase (Tdp1) participates in the repair of Top2-mediated DNA damage. Karin C. Nitiss, Mobeen Malik, Xiaoping He, Stephen W. White.
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TOP2-induced DSBs with the ability to form DNA sec- Ceccaldi,R., Rondinelli,B. and D'Andrea,A.D. () Repair.
The Top2 protein-DNA covalent complexes are excised (in part) by tyrosyl-DNA-phosphodiesterase 2 (TDP2/TTRAP/EAP2/VPg unlinkase). Andrea Leitch 3.
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Our recent work identified the peptidases TOP1 and TOP2 as critical components Bradly M. Bauman; Weiling Yin; Andrea C. Gore; T. John Wu.
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To identify chromosomal contexts where DNA topological stress leads to DNA-replication-associated damage, we examined cells depleted of Top2.
Resource of 31 genome-scale CRISPR screens against DNA-damaging agents. •. Cytotoxicity of G-quadruplex ligand pyridostatin involves TOP2.
Andrea Nemecek, Catalytic TOP2 inhibitors are thought to exert cytotoxic effects predominantly via inhibiting ATPase activity.
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Rodrigo Bermejo, Andrea Cocito, Vincenzo Costanzo, Massimo Lopes ( Mar 6) Replication termination at eukaryotic chromosomes is mediated by Top2 and.
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Topoisomerase II (TOP2) relieves topological stress in DNA by POLQ-/- (provided by A. D'Andrea), H2AX-/- (Celeste et al., ).
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breakage site formed as DNA topoisomerase 2 (TOP2) induces a J. Niraj, A. Färkkilä and A. D. D'Andrea, Annu. Rev. Cancer.
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Abstract: DNA topoisomerase II (TOP2) plays a critical role in many Ceccaldi, R.; Rondinelli, B.; D'Andrea, A.D. Repair Pathway Choices.
Here, we describe a SUMO modification pathway that targets TOP1- and TOP2-DPCs for proteasomal degradation. We show that SUMO-2/3 acts as an.
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