Do you have Crohn's? Believe me, I know what that's like. This 100+ page eBook guide contains everything I did to beat it, and the tools you need overcome it too. Don’t Miss These Popular Posts Epsom Salt: Why You Need It and How to Use ItWhat’s with the Craving? Causes of Food Cravings RevealedEssential Oils: Fad or Fantastic?A Gentle Vaccine Detox For ChildrenDosing & UsesDosage Forms & Strengths Initial: 1 mEq/kg/dose IV x1; base subsequent doses on results of arterial blood pH and PaCO2 as well as calculation of base deficit Repeat doses may be considered in the setting of prolonged cardiac arrest only after adequate alveolar ventilation has been established 50 mEq IV over 5 minutes 2-5 mEq/kg IV infusion over 4-8 hr depending on the severity of acidosis as judged by the lowering of total CO2 content, clinical condition and pH Severe Metabolic Acidosis (Except Hypercarbic Acidosis) 90 to 180 mEq/L (~ 7.5-15 g) at a rate of 1-1.5 L (first hour);
adjust for further management as needed Dosage Forms & Strengths Infants, <2 years (use 4.2% solution) Initial: 1 mEq/kg/min given over 1-2 minutes IV/IO, THEN 1 mEq/kg IV q10min of arrest Not to exceed 8 mEq/kg/day >2 years Older children: 2-5 mEq/kg IV infusion over 4-8 hr depending on the severity of acidosis as judged by the lowering of total CO2 content, clinical condition and pH InteractionsInteraction CheckerEnter a drug nameNo Results ContraindicatedSerious - Use AlternativeSignificant - Monitor CloselyMinorSeverityName Hypersensitivity, metabolic or respiratory alkalosis, hypocalcemia, excessive Cl- loss from vomiting or GI suctioning Patients at risk of developing diuretic-induced hypochloremic alkalosis Not first-line for resuscitation Edematous or Na-retaining states, history of CHF, renal impairment, cirrhosis, HTN, children with DKA, concurrent corticosteroid use Use caution in patients with cirrhosis, heart failure, renal impairment, peptic ulcer disease, or edema
Avoid extravasation (may cause chemical cellulitis, tissue necrosis, ulceration & sloughing due to alkalinity) Lactation: Not known if excreted in breast milkControlled studies in pregnant women show no evidence of fetal risk.Either animal studies show no risk but human studies not available or animal studies showed minor risks and human studies done and showed no risk.C:Use with caution if benefits outweigh risks. Animal studies show risk and human studies not available or neither animal nor human studies done.D:Use in LIFE-THREATENING emergencies when no safer drug available. Positive evidence of human fetal risk.X:Do not use in pregnancy. Risks involved outweigh potential benefits. Bicarbonate reacts with H+ ions to form water & carbon dioxide. It acts as a buffer against acidosis by raising blood pH Onset: 15 min (IV) Duration: 1-2 hr (IV); Therapeutic range: 24-31 mEq/L Absorption: Well absorbed orally Additive: ascorbic acid, carboplatin, carmustine, ciprofloxacin, cisplatin, dobutamine, dopamine, epinephrine, hydromorphone, imipenem-cilastatin, labetalol, meperidine (?), meropenem, morphine, norepinephrine, penicillin G potassium, pentazocine, pentobarbital, procaine, sodium lactate, streptomycin, succinylcholine, vancomycin(?), vit B/C
Syringe: bupivacaine(?), epinephrine(?), etidocaine, glycopyrrolate, lidocaine(?), mepivacaine, metoclopramide, thiopental Y-site: allopurinol, amiodarone, ampho B cholesterylSO4, CaCl2, Ca gluconate, ciprofloxacin(?), cisatarcurium(?), doxorubicin liposomal, fenoldopam, hetastarch, idarubicin, imipenem-cilastatin, inamrinone, leucovorin, midazolam, nalbuphine, ondansetron, oxacillin, sargramostim, verapamil, vincristine, vindesine, vinorelbine Not spec: diazepam, tetracycline Additive: aminophylline, ampho B, atropine, Ca gluconate, clindamycin, erythromycin, heparin, hydrocortisone, KCl, verapamil Y-site: heparin, morphine, KCl, vancomycin, vit B/C Administered IV, either undiluted or diluted in other fluids, or By SC if diluted to isotonicity Store at controlled room temperature Protect from freezing & temp >40°C Do not use if unclear or contains a precipitate FormularyAdding plans allows you to compare formulary status to other drugs in the same class.
To view formulary information first create a list of plans. Your list will be saved and can be edited at any time.Create Your List of PlansAdding plans allows you to:View the formulary and any restrictions for each plan.pare formulary status to other drugs in the same class.Access your plan list on any device – mobile or desktop. The above information is provided for general informational and educational purposes only. Individual plans may vary and formulary information changes. Contact the applicable plan provider for the most current information. This drug is available at the lowest co-pay. commonly, these are generic drugs. This drug is available at a middle level co-pay. commonly, these are "preferred" (on formulary) brand drugs. This drug is available at a higher level co-pay. commonly, these are "non-preferred" brand drugs. commonly, these are "non-preferred" brand drugs or specialty NOT COVERED – Drugs that are not covered by the plan.
Prior Authorization Drugs thatThis restriction requires that specific clinical criteria be met prior to the approval of the Quantity Limits Drugs that have quantity limits associated with each prescription. restriction typically limits the quantity of the drug that will Step Therapy Drugs that have step therapy associated with each prescription. typically requires that certain criteria be met prior to approval for the prescription. Other Restrictions Drugs that have restrictions other than prior authorization, quantity limits, and step therapy associated with each prescription. The recipient will receive more details and instructions to access this offer. By clicking send, you acknowledge that you have permission to email the recipient with this information. Diabetic Ketoacidosis (DKA) - Topic Overview What is diabetic ketoacidosis (DKA)? Diabetic ketoacidosis (DKA) is a life-threatening condition that develops when cells in the body are unable to get the sugar (glucose) they need for energy because there is not enough insulin.
When the sugar cannot get into the cells, it stays in the blood. The kidneys filter some of the sugar from the blood and remove it from the body through urine. Because the cells cannot receive sugar for energy, the body begins to break down fat and muscle for energy. When this happens, ketones, or fatty acids, are produced and enter the bloodstream, causing the chemical imbalance (metabolic acidosis) called diabetic ketoacidosis. Ketoacidosis can be caused by not getting enough insulin, having a severe infection or other illness, becoming severely dehydrated, or some combination of these things. It can occur in people who have little or no insulin in their bodies (mostly people with type 1 diabetes but it can happen with type 2 diabetes, especially children) when their blood sugar levels are high. What are the symptoms? Your blood sugar may be quite high before you notice symptoms, which include: Flushed, hot, dry skin. Feeling thirsty and urinating a lot.
Drowsiness or difficulty waking up. Young children may lack interest in their normal activities. A strong, fruity breath odor. Loss of appetite, belly pain, and vomiting. How is DKA diagnosed? Laboratory tests, including blood and urine tests, are used to confirm a diagnosis of diabetic ketoacidosis. Tests for ketones are available for home use. Keep some test strips nearby in case your blood sugar level becomes high. How is it treated? When ketoacidosis is severe, it must be treated in the hospital, often in an intensive care unit. Treatment involves giving insulin and fluids through your vein and closely watching certain chemicals in your blood (electrolytes). The doctors and nurses will watch you closely to be sure that your brain does not swell as the fluids treat your dehydration. It can take several days for your blood sugar level to return to a target range. How can I prevent DKA? The risk for DKA is higher when you are sick. Stress hormones released due to illness can raise your blood sugar.